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Blood Advances|July 1, 2025
Impact of transplant conditioning, NPM1 mutations, and measurable residual disease in FLT3-ITD acute myeloid leukemiaMark J Levis, Mehdi Hamadani, Brent R Logan, et al.Blood|January 8, 2025
Measurable residual disease and posttransplantation gilteritinib maintenance for patients with FLT3-ITD-mutated AMLMark J Levis, Mehdi Hamadani, Brent R Logan, et al.Cell|May 7, 2024
Mapping the cellular biogeography of human bone marrow niches using single-cell transcriptomics and proteomic imagingShovik Bandyopadhyay, Michael P Duffy, Kyung Jin Ahn, et al.Journal for Immunotherapy of Cancer|May 22, 2023
Intrinsic suppression of type I interferon production underlies the therapeutic efficacy of IL-15-producing natural killer cells in B-cell acute lymphoblastic leukemiaAnil Kumar, Adeleh Taghi Khani, Caroline Duault, et al.Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|March 12, 2024
Gilteritinib as Post-Transplant Maintenance for AML With Internal Tandem Duplication Mutation of FLT3Mark J Levis, Mehdi Hamadani, Brent Logan, et al.Cancer Discovery|May 12, 2019
Rational Targeting of Cooperating Layers of the Epigenome Yields Enhanced Therapeutic Efficacy against AMLCihangir Duy, Matt Teater, Francine E Garrett-Bakelman, et al.Cancer Discovery|September 2, 2022
Small-Molecule Inhibition of the Acyl-Lysine Reader ENL as a Strategy against Acute Myeloid LeukemiaYiman Liu, Qinglan Li, Fatemeh Alikarami, et al.Leukemia|July 9, 2025
Chemoresistance of TP53 mutant acute myeloid leukemia requires the mevalonate byproduct, geranylgeranyl pyrophosphate, for induction of an adaptive stress responseSarah J Skuli, A'ishah Bakayoko, Marisa Kruidenier, et al.Cancer Discovery|April 19, 2017
Chemotherapy-Resistant Human Acute Myeloid Leukemia Cells Are Not Enriched for Leukemic Stem Cells but Require Oxidative MetabolismThomas Farge, Estelle Saland, Fabienne de Toni, et al.Blood|May 1, 2020
Human erythroleukemia genetics and transcriptomes identify master transcription factors as functional disease driversAlexandre Fagnan, Frederik Otzen Bagger, Maria-Riera Piqué-Borràs, et al.Pageof 19