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Nucleic Acids Research|March 21, 2025
Light-induced expression of gRNA allows for optogenetic gene editing of T lymphocytes in vivoDiego Velasquez Pulgarin, Nathalie Pelo, Lin Ferrandiz, et al.
Biorxiv : the Preprint Server for Biology|November 21, 2023
Light induced expression of gRNA allows for optogenetic gene editing of T lymphocytes in vivoDiego Velasquez Pulgarin, Nathalie Pelo, Lin Ferrandiz, et al.
Plos One|July 30, 2010
Self protection from anti-viral responses--Ro52 promotes degradation of the transcription factor IRF7 downstream of the viral Toll-Like receptorsRowan Higgs, Elisa Lazzari, Claire Wynne, et al.
Experimental Dermatology|December 13, 2016
TRIM21 is important in the early phase of inflammation in the imiquimod-induced psoriasis-like skin inflammation mouse modelHanne Vinter, Ane Langkilde, Vijole Ottosson, et al.
EMBO Reports|November 7, 2022
The bromodomain protein TRIM28 controls the balance between growth and invasiveness in melanomaWilliam A Nyberg, Diego A Velasquez-Pulgarin, Tianlin He, et al.
The Journal of Biological Chemistry|October 29, 2025
Mapping the intracellular HMGB1 interactome and alterations induced by Toll-like receptor 4 activationRebecka Heinbäck, Marit van der Zijde, Choi Har Tsang, et al.
Journal of Immunology (Baltimore, Md. : 1950)|August 27, 2013
Expression of the immune regulator tripartite-motif 21 is controlled by IFN regulatory factorsMaria Sjöstrand, Aurélie Ambrosi, Susanna Brauner, et al.
Multiple Sclerosis (Houndmills, Basingstoke, England)|April 1, 2010
Interleukin 18 receptor 1 expression distinguishes patients with multiple sclerosisAlan Gillett, Melanie Thessen Hedreul, Mohsen Khademi, et al.
The Journal of Biological Chemistry|August 25, 2011
Anti-Ro52 autoantibodies from patients with Sjögren's syndrome inhibit the Ro52 E3 ligase activity by blocking the E3/E2 interfaceAlexander Espinosa, Janosch Hennig, Aurélie Ambrosi, et al.
Arthritis & Rheumatology (Hoboken, N.J.)|June 29, 2026
DNA (cytosine-5)-methyltransferase 3A (Dnmt3a) mutations limit normal and autoreactive CD4+ T follicular helper responses and attenuate T cell-driven joint inflammationYunbing Shen, Sanjaykumar Boddul, Zhaojun Li, et al.
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