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Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|October 15, 2019
YAP and TAZ Mediate Osteocyte Perilacunar/Canalicular RemodelingChristopher D Kegelman, Jennifer C Coulombe, Kelsey M Jordan, et al.
Bone|September 3, 2023
Loss of Nmp4 enhances bone gain from sclerostin antibody administrationCrystal Korff, Michele Adaway, Emily G Atkinson, et al.
Bone|February 8, 2013
Reduced gravitational loading does not account for the skeletal effect of botulinum toxin-induced muscle inhibition suggesting a direct effect of muscle on boneStuart J Warden, Matthew R Galley, Jeffrey S Richard, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|February 7, 2018
Skeletal cell YAP and TAZ combinatorially promote bone developmentChristopher D Kegelman, Devon E Mason, James H Dawahare, et al.
Development (Cambridge, England)|November 29, 2017
The skeletal phenotype of achondrogenesis type 1A is caused exclusively by cartilage defectsIan M Bird, Susie H Kim, Devin K Schweppe, et al.
Bone|September 3, 2018
Vhl deficiency in osteocytes produces high bone mass and hematopoietic defectsGabriela G Loots, Alexander G Robling, Jiun C Chang, et al.
Bone|November 15, 2011
Sost downregulation and local Wnt signaling are required for the osteogenic response to mechanical loadingXiaolin Tu, Yumie Rhee, Keith W Condon, et al.
Human Molecular Genetics|December 20, 2007
Rac1 mediates the osteoclast gains-in-function induced by haploinsufficiency of Nf1Jincheng Yan, Shi Chen, Yingze Zhang, et al.
Science Translational Medicine|November 15, 2013
Sclerostin inhibition reverses skeletal fragility in an Lrp5-deficient mouse model of OPPG syndromeRajendra Kedlaya, Shreya Veera, Daniel J Horan, et al.
JCI Insight|September 8, 2017
Old age causes de novo intracortical bone remodeling and porosity in miceMarilina Piemontese, Maria Almeida, Alexander G Robling, et al.
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