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Alexander G Robling

Showing results (81-90 of 136) with videos related to

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Bone|June 15, 2016
Enhanced Wnt signaling improves bone mass and strength, but not brittleness, in the Col1a1(+/mov13) mouse model of type I Osteogenesis ImperfectaChristina M Jacobsen, Marissa A Schwartz, Heather J Roberts, et al.
Journal of Cellular Physiology|January 19, 2006
HMGB1 expression and release by bone cellsKanokwan Charoonpatrapong, Rita Shah, Alexander G Robling, et al.
Journal of Cellular Physiology|July 7, 2011
Immortalization and characterization of osteoblast cell lines generated from wild-type and Nmp4-null mouse bone marrow stromal cells using murine telomerase reverse transcriptase (mTERT)Marta B Alvarez, Paul Childress, Binu K Philip, et al.
Journal of Bone and Mineral Metabolism|August 1, 2018
Finite-element analysis of the mouse proximal ulna in response to elbow loadingFeifei Jiang, Aydin Jalali, Chie Deguchi, et al.
Iscience|October 5, 2019
Lrp4 Mediates Bone Homeostasis and Mechanotransduction through Interaction with Sclerostin In VivoWhitney A Bullock, April M Hoggatt, Daniel J Horan, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|October 15, 2019
YAP and TAZ Mediate Osteocyte Perilacunar/Canalicular RemodelingChristopher D Kegelman, Jennifer C Coulombe, Kelsey M Jordan, et al.
Bone|September 3, 2023
Loss of Nmp4 enhances bone gain from sclerostin antibody administrationCrystal Korff, Michele Adaway, Emily G Atkinson, et al.
Bone|February 8, 2013
Reduced gravitational loading does not account for the skeletal effect of botulinum toxin-induced muscle inhibition suggesting a direct effect of muscle on boneStuart J Warden, Matthew R Galley, Jeffrey S Richard, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|February 7, 2018
Skeletal cell YAP and TAZ combinatorially promote bone developmentChristopher D Kegelman, Devon E Mason, James H Dawahare, et al.
Development (Cambridge, England)|November 29, 2017
The skeletal phenotype of achondrogenesis type 1A is caused exclusively by cartilage defectsIan M Bird, Susie H Kim, Devin K Schweppe, et al.
Pageof 14

Showing results (81-90 of 136) with videos related to

Sort By:
Pageof 14
Bone|June 15, 2016
Enhanced Wnt signaling improves bone mass and strength, but not brittleness, in the Col1a1(+/mov13) mouse model of type I Osteogenesis ImperfectaChristina M Jacobsen, Marissa A Schwartz, Heather J Roberts, et al.
Journal of Cellular Physiology|January 19, 2006
HMGB1 expression and release by bone cellsKanokwan Charoonpatrapong, Rita Shah, Alexander G Robling, et al.
Journal of Cellular Physiology|July 7, 2011
Immortalization and characterization of osteoblast cell lines generated from wild-type and Nmp4-null mouse bone marrow stromal cells using murine telomerase reverse transcriptase (mTERT)Marta B Alvarez, Paul Childress, Binu K Philip, et al.
Journal of Bone and Mineral Metabolism|August 1, 2018
Finite-element analysis of the mouse proximal ulna in response to elbow loadingFeifei Jiang, Aydin Jalali, Chie Deguchi, et al.
Iscience|October 5, 2019
Lrp4 Mediates Bone Homeostasis and Mechanotransduction through Interaction with Sclerostin In VivoWhitney A Bullock, April M Hoggatt, Daniel J Horan, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|October 15, 2019
YAP and TAZ Mediate Osteocyte Perilacunar/Canalicular RemodelingChristopher D Kegelman, Jennifer C Coulombe, Kelsey M Jordan, et al.
Bone|September 3, 2023
Loss of Nmp4 enhances bone gain from sclerostin antibody administrationCrystal Korff, Michele Adaway, Emily G Atkinson, et al.
Bone|February 8, 2013
Reduced gravitational loading does not account for the skeletal effect of botulinum toxin-induced muscle inhibition suggesting a direct effect of muscle on boneStuart J Warden, Matthew R Galley, Jeffrey S Richard, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|February 7, 2018
Skeletal cell YAP and TAZ combinatorially promote bone developmentChristopher D Kegelman, Devon E Mason, James H Dawahare, et al.
Development (Cambridge, England)|November 29, 2017
The skeletal phenotype of achondrogenesis type 1A is caused exclusively by cartilage defectsIan M Bird, Susie H Kim, Devin K Schweppe, et al.
Pageof 14