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Journal of Medicinal Chemistry|January 20, 2006
Structure-activity study of brassinin derivatives as indoleamine 2,3-dioxygenase inhibitorsPaul Gaspari, Tinku Banerjee, William P Malachowski, et al.Cancer Research|January 11, 2007
Bin1 ablation in mammary gland delays tissue remodeling and drives cancer progressionMee Young Chang, Janette Boulden, Erika Sutanto-Ward, et al.Journal of Immunology (Baltimore, Md. : 1950)|December 30, 2021
The Immunomodulatory Enzyme IDO2 Mediates Autoimmune Arthritis through a Nonenzymatic MechanismLauren M F Merlo, Weidan Peng, James B DuHadaway, et al.Journal of Medicinal Chemistry|July 31, 2008
Structure based development of phenylimidazole-derived inhibitors of indoleamine 2,3-dioxygenaseSanjeev Kumar, Daniel Jaller, Bhumika Patel, et al.Oncogene|June 6, 2003
Transformation-selective apoptotic program triggered by farnesyltransferase inhibitors requires Bin1James B DuHadaway, Wei Du, Scott Donover, et al.Cancer Research|August 19, 2007
Bin1 ablation increases susceptibility to cancer during aging, particularly lung cancerMee Young Chang, Janette Boulden, Jessica B Katz, et al.Journal for Immunotherapy of Cancer|May 30, 2025
Neovascular pruning by IDO1 inhibitors can potentiate immunogenic cytotoxicity of ischemia-targeted agents to synergistically enhance anti-PD-1 responsivenessShih-Chun Shen, Souvik Dey, James B DuHadaway, et al.Journal of Medicinal Chemistry|March 6, 2008
Indoleamine 2,3-dioxygenase is the anticancer target for a novel series of potent naphthoquinone-based inhibitorsSanjeev Kumar, William P Malachowski, James B DuHadaway, et al.Cancer Research|January 20, 2007
Inhibition of indoleamine 2,3-dioxygenase in dendritic cells by stereoisomers of 1-methyl-tryptophan correlates with antitumor responsesDe-Yan Hou, Alexander J Muller, Madhav D Sharma, et al.Cancer Immunology Research|February 24, 2021
IDO1 Signaling through GCN2 in a Subpopulation of Gr-1<sup>+</sup> Cells Shifts the IFNγ/IL6 Balance to Promote NeovascularizationSouvik Dey, Arpita Mondal, James B DuHadaway, et al.Pageof 6