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Cancer Biology & Therapy|December 14, 2011
Cardiac and gastrointestinal liabilities caused by deficiency in the immune modulatory enzyme indoleamine 2,3-dioxygenaseMee Young Chang, Courtney Smith, James B DuHadaway, et al.
Journal of the American College of Surgeons|February 10, 2018
A Sub-Type of Familial Pancreatic Cancer: Evidence and Implications of Loss-of-Function Polymorphisms in Indoleamine-2,3-Dioxygenase-2Avinoam Nevler, Alexander J Muller, Joseph A Cozzitorto, et al.
Cancer Immunology, Immunotherapy : CII|July 20, 2010
Non-hematopoietic expression of IDO is integrally required for inflammatory tumor promotionAlexander J Muller, James B DuHadaway, Mee Young Chang, et al.
Frontiers in Immunology|September 25, 2020
Differential Roles of IDO1 and IDO2 in T and B Cell Inflammatory Immune ResponsesLauren M F Merlo, James B DuHadaway, James D Montgomery, et al.
European Journal of Medicinal Chemistry|November 24, 2018
Diaryl hydroxylamines as pan or dual inhibitors of indoleamine 2,3-dioxygenase-1, indoleamine 2,3-dioxygenase-2 and tryptophan dioxygenaseMaria Winters, James B DuHadaway, Khoa N Pham, et al.
Journal of Cellular Biochemistry|May 6, 2015
Cardiac-specific disruption of Bin1 in mice enables a model of stress- and age-associated dilated cardiomyopathyLisa D Laury-Kleintop, Jennifer R Mulgrew, Ido Heletz, et al.
European Journal of Medicinal Chemistry|December 31, 2015
O-alkylhydroxylamines as rationally-designed mechanism-based inhibitors of indoleamine 2,3-dioxygenase-1William P Malachowski, Maria Winters, James B DuHadaway, et al.
Cancer Research|March 15, 2008
Bin3 deletion causes cataracts and increased susceptibility to lymphoma during agingArivudainambi Ramalingam, James B Duhadaway, Erika Sutanto-Ward, et al.
International Immunology|January 10, 2014
IDO2 is critical for IDO1-mediated T-cell regulation and exerts a non-redundant function in inflammationRichard Metz, Courtney Smith, James B DuHadaway, et al.
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