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Proceedings of the National Academy of Sciences of the United States of America|May 25, 2005
Increased glutathione S-transferase activity rescues dopaminergic neuron loss in a Drosophila model of Parkinson's diseaseAlexander J Whitworth, Dorothy A Theodore, Jessica C Greene, et al.
Plos Genetics|November 21, 2014
The complex I subunit NDUFA10 selectively rescues Drosophila pink1 mutants through a mechanism independent of mitophagyJoe H Pogson, Rachael M Ivatt, Alvaro Sanchez-Martinez, et al.
Scientific Reports|August 20, 2016
Parkinson disease-linked GBA mutation effects reversed by molecular chaperones in human cell and fly modelsAlvaro Sanchez-Martinez, Michelle Beavan, Matthew E Gegg, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 31, 2008
The PINK1/Parkin pathway regulates mitochondrial morphologyAngela C Poole, Ruth E Thomas, Laurie A Andrews, et al.
The Journal of Cell Biology|March 4, 2018
Basal mitophagy is widespread in <i>Drosophila</i> but minimally affected by loss of Pink1 or parkinJuliette J Lee, Alvaro Sanchez-Martinez, Aitor Martinez Zarate, et al.
Human Molecular Genetics|August 15, 2014
Mitochondrial defects and neuromuscular degeneration caused by altered expression of Drosophila Gdap1: implications for the Charcot-Marie-Tooth neuropathyVíctor López Del Amo, Marta Seco-Cervera, José Luís García-Giménez, et al.
Plos One|September 7, 2012
Modeling pathogenic mutations of human twinkle in Drosophila suggests an apoptosis role in response to mitochondrial defectsAlvaro Sanchez-Martinez, Manuel Calleja, Susana Peralta, et al.
The Journal of Biological Chemistry|March 9, 2016
Superoxide Dismutase (SOD)-mimetic M40403 Is Protective in Cell and Fly Models of Paraquat Toxicity: IMPLICATIONS FOR PARKINSON DISEASERoberta Filograna, Vinay K Godena, Alvaro Sanchez-Martinez, et al.
Cells|May 16, 2023
USP8 Down-Regulation Promotes Parkin-Independent Mitophagy in the <i>Drosophila</i> Brain and in Human NeuronsSofia Mauri, Greta Bernardo, Aitor Martinez, et al.
Plos Genetics|July 7, 2025
Clu1/Clu form mitochondria-associated granules upon metabolic transitions and regulate mitochondrial protein translation via ribosome interactionsLeonor Miller-Fleming, Wing Hei Au, Laura Raik, et al.
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