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Cell Death and Differentiation|January 21, 2017
Loss of menin in osteoblast lineage affects osteocyte-osteoclast crosstalk causing osteoporosisPeng Liu, Sooyeon Lee, Jeanette Knoll, et al.
Nature Metabolism|February 23, 2021
Genome-wide discovery of genetic loci that uncouple excess adiposity from its comorbiditiesLam O Huang, Alexander Rauch, Eugenia Mazzaferro, et al.
Nature Genetics|March 6, 2019
Osteogenesis depends on commissioning of a network of stem cell transcription factors that act as repressors of adipogenesisAlexander Rauch, Anders K Haakonsson, Jesper G S Madsen, et al.
Bone Research|April 1, 2025
KIAA1199 (CEMIP) regulates adipogenesis and whole-body energy metabolismLi Chen, Kaikai Shi, Nicholas Ditzel, et al.
Nature Genetics|March 27, 2019
Author Correction: Osteogenesis depends on commissioning of a network of stem cell transcription factors that act as repressors of adipogenesisAlexander Rauch, Anders K Haakonsson, Jesper G S Madsen, et al.
European Journal of Endocrinology|February 7, 2023
GIP reduces osteoclast activity and improves osteoblast survival in primary human bone cellsMorten S Hansen, Kent Søe, Line L Christensen, et al.
Bone Research|June 14, 2026
An ITGA11 expressing subpopulation as predictor for the donor-specific osteogenic capacity of stromal cellsAli Jasim Mohammad Jamil, Kaja Madsen, Souad Daamouch, et al.
Nature Medicine|September 12, 2025
Genetic subtyping of obesity reveals biological insights into the uncoupling of adiposity from its cardiometabolic comorbiditiesNathalie Chami, Zhe Wang, Victor Svenstrup, et al.
Medrxiv : the Preprint Server for Health Sciences|March 10, 2025
Genetic subtyping of obesity reveals biological insights into the uncoupling of adiposity from its cardiometabolic comorbiditiesNathalie Chami, Zhe Wang, Victor Svenstrup, et al.
Cell Metabolism|June 4, 2010
Glucocorticoids suppress bone formation by attenuating osteoblast differentiation via the monomeric glucocorticoid receptorAlexander Rauch, Sebastian Seitz, Ulrike Baschant, et al.
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