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Endocrinology|February 12, 2005
Thioredoxin-interacting protein is stimulated by glucose through a carbohydrate response element and induces beta-cell apoptosisAlexandra H Minn, Christian Hafele, Anath Shalev
Biochemistry|September 13, 2006
Metabolism-independent sugar effects on gene transcription: the role of 3-O-methylglucoseAlexandra H Minn, Francesca M Couto, Anath Shalev
Biochemical and Biophysical Research Communications|June 20, 2006
Exenatide inhibits beta-cell apoptosis by decreasing thioredoxin-interacting proteinJunqin Chen, Francesca M Couto, Alexandra H Minn, et al.
Lancet (London, England)|April 9, 2004
Insulinomas and expression of an insulin splice variantAlexandra H Minn, Mark Kayton, Dominique Lorang, et al.
Biochemical and Biophysical Research Communications|September 7, 2005
Gene expression profiling in INS-1 cells overexpressing thioredoxin-interacting proteinAlexandra H Minn, Cynthia A Pise-Masison, Michael Radonovich, et al.
Molecular Endocrinology (Baltimore, Md.)|November 20, 2004
Increased insulin translation from an insulin splice-variant overexpressed in diabetes, obesity, and insulin resistanceAlexandra H Minn, Hong Lan, Mary E Rabaglia, et al.
Biochemical and Biophysical Research Communications|October 3, 2003
Resistin is expressed in pancreatic isletsAlexandra H Minn, Noelle B Patterson, Stephanie Pack, et al.
Metabolism: Clinical and Experimental|June 16, 2007
Exenatide blocks JAK1-STAT1 in pancreatic beta cellsFrancesca M Couto, Alexandra H Minn, Cynthia A Pise-Masison, et al.
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