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Journal of Cell Science|October 23, 2019
Adaptor protein complexes and disease at a glanceAnneri Sanger, Jennifer Hirst, Alexandra K Davies, et al.Toxicological Sciences : an Official Journal of the Society of Toxicology|January 15, 2017
From the Cover: Does the Assessment of Nondisjunction Provide a More Sensitive Assay for the Detection of Aneugens?Joanne M Elloway, Alexandra K Davies, Julie E Hayes, et al.The Journal of Cell Biology|April 5, 2024
The role of the AP-1 adaptor complex in outgoing and incoming membrane trafficMargaret S Robinson, Robin Antrobus, Anneri Sanger, et al.Nature Communications|August 29, 2023
Deep and fast label-free Dynamic Organellar MappingJulia P Schessner, Vincent Albrecht, Alexandra K Davies, et al.Biorxiv : the Preprint Server for Biology|March 3, 2025
Arrayed CRISPR/Cas9 Loss-Of-Function Screen in a Neuronal Model of Adaptor Protein Complex 4 Deficiency Identifies Modulators of ATG9A TraffickingMarvin Ziegler, Cedric Böger, Julian E Alecu, et al.Scientific Reports|February 7, 2018
Clathrin heavy chain 22 contributes to the control of neuropeptide degradation and secretion during neuronal developmentMichael S Nahorski, Georg H H Borner, Samiha S Shaikh, et al.JCI Insight|May 19, 2026
CRISPR/Cas9 loss-of-function screen in a neuronal model of AP-4 deficiency identifies ATG9A trafficking modulatorsMarvin Ziegler, Cedric Günter, Julian E Alecu, et al.Traffic (Copenhagen, Denmark)|January 13, 2016
Molecular Basis for the Interaction Between AP4 β4 and its Accessory Protein, TepsinMeredith N Frazier, Alexandra K Davies, Markus Voehler, et al.Nature Communications|September 29, 2018
AP-4 vesicles contribute to spatial control of autophagy via RUSC-dependent peripheral delivery of ATG9AAlexandra K Davies, Daniel N Itzhak, James R Edgar, et al.Nature Communications|February 26, 2022
AP-4-mediated axonal transport controls endocannabinoid production in neuronsAlexandra K Davies, Julian E Alecu, Marvin Ziegler, et al.Pageof 2