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Proceedings of the National Academy of Sciences of the United States of America|January 19, 2017
Myofibril breakdown during atrophy is a delayed response requiring the transcription factor PAX4 and desmin depolymerizationAlexandra Volodin, Idit Kosti, Alfred Lewis Goldberg, et al.The Journal of Cell Biology|August 1, 2018
GSK3-β promotes calpain-1-mediated desmin filament depolymerization and myofibril loss in atrophyDina Aweida, Inga Rudesky, Alexandra Volodin, et al.Nature Communications|March 15, 2020
A signaling hub of insulin receptor, dystrophin glycoprotein complex and plakoglobin regulates muscle sizeYara Eid Mutlak, Dina Aweida, Alexandra Volodin, et al.Cancers|October 21, 2020
Tumor Treating Fields (TTFields) Hinder Cancer Cell Motility through Regulation of Microtubule and Acting DynamicsTali Voloshin, Rosa Sara Schneiderman, Alexandra Volodin, et al.Biomolecules|October 27, 2022
Tumor Treating Fields (TTFields) Reversibly Permeabilize the Blood-Brain Barrier In Vitro and In VivoEllaine Salvador, Almuth F Kessler, Dominik Domröse, et al.International Journal of Molecular Sciences|November 26, 2022
Tumor Treating Fields (TTFields) Concomitant with Immune Checkpoint Inhibitors Are Therapeutically Effective in Non-Small Cell Lung Cancer (NSCLC) In Vivo ModelYiftah Barsheshet, Tali Voloshin, Boris Brant, et al.Molecular Cancer Therapeutics|June 30, 2025
Cancer Cell Permeability Induced by Tumor Treating Fields (TTFields) as a Physical Approach to Improve Chemotherapy Uptake and Overcome Multidrug ResistanceBella Koltun, Tali Voloshin, Cfir David, et al.Cell Death & Disease|March 28, 2025
Role of the PI3K/AKT signaling pathway in the cellular response to Tumor Treating Fields (TTFields)Anat Klein-Goldberg, Tali Voloshin, Efrat Zemer Tov, et al.Pageof 1