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Neuroscience Letters|June 14, 2020
Collagens at the vertebrate neuromuscular junction, from structure to pathologiesClaire Legay, Alexandre Dobbertin
Molecular and Cellular Neurosciences|March 4, 2008
Identification of cis-acting elements involved in acetylcholinesterase RNA alternative splicingManuel Guerra, Alexandre Dobbertin, Claire Legay
Chemico-Biological Interactions|October 24, 2012
Developmental consequences of the ColQ/MuSK interactionsJennifer Karmouch, Alexandre Dobbertin, Severine Sigoillot, et al.
Advances in Experimental Medicine and Biology|September 8, 2006
DSD-1-Proteoglycan/Phosphacan and receptor protein tyrosine phosphatase-beta isoforms during development and regeneration of neural tissuesAndreas Faissner, Nicolas Heck, Alexandre Dobbertin, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 10, 2009
Targeting of acetylcholinesterase in neurons in vivo: a dual processing function for the proline-rich membrane anchor subunit and the attachment domain on the catalytic subunitAlexandre Dobbertin, Anna Hrabovska, Korami Dembele, et al.
Anesthesiology|September 30, 2005
Cholinesterases and the resistance of the mouse diaphragm to the effect of tubocurarineTu Nguyen-Huu, Alexandre Dobbertin, Julien Barbier, et al.
The European Journal of Neuroscience|November 19, 2004
The extracellular matrix glycoprotein Tenascin-C is expressed by oligodendrocyte precursor cells and required for the regulation of maturation rate, survival and responsiveness to platelet-derived growth factorJeremy Garwood, Emmanuel Garcion, Alexandre Dobbertin, et al.
Molecular and Cellular Neurosciences|August 11, 2007
Evidence for distinct leptomeningeal cell-dependent paracrine and EGF-linked autocrine regulatory pathways for suppression of fibrillar collagens in astrocytesNicolas Heck, Jeremy Garwood, Alexandre Dobbertin, et al.
Molecular and Cellular Neurosciences|December 31, 2003
Regulation of RPTPbeta/phosphacan expression and glycosaminoglycan epitopes in injured brain and cytokine-treated gliaAlexandre Dobbertin, Kate E Rhodes, Jeremy Garwood, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|March 20, 2016
Neuromuscular junction immaturity and muscle atrophy are hallmarks of the ColQ-deficient mouse, a model of congenital myasthenic syndrome with acetylcholinesterase deficiencySéverine M Sigoillot, Francine Bourgeois, Jennifer Karmouch, et al.
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