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Plos One|January 18, 2013
Automated filtering of intrinsic movement artifacts during two-photon intravital microscopyDenis Soulet, Alexandre Paré, Julien Coste, et al.
Brain, Behavior, and Immunity|July 20, 2016
Involvement of the IL-1 system in experimental autoimmune encephalomyelitis and multiple sclerosis: Breaking the vicious cycle between IL-1β and GM-CSFAlexandre Paré, Benoit Mailhot, Sébastien A Lévesque, et al.
Blood|May 1, 2012
Platelets can enhance vascular permeabilityNathalie Cloutier, Alexandre Paré, Richard W Farndale, et al.
European Journal of Immunology|October 6, 2017
Ly6Chigh monocytes facilitate transport of Murid herpesvirus 68 into inflamed joints of arthritic miceManon LeBel, Benoit Egarnes, Alexandre Brunet, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 7, 2026
Inflammatory monocyte-derived macrophages drive pain via their production of nerve growth factor after peripheral nerve injury in miceMaxime Kusik, Alexandre Paré, Felipe Da Gama Monteiro, et al.
International Journal of Molecular Sciences|December 23, 2022
The J Domain of Sacsin Disrupts Intermediate Filament AssemblyAfrooz Dabbaghizadeh, Alexandre Paré, Zacharie Cheng-Boivin, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 24, 2018
IL-1β enables CNS access to CCR2hi monocytes and the generation of pathogenic cells through GM-CSF released by CNS endothelial cellsAlexandre Paré, Benoit Mailhot, Sébastien A Lévesque, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 23, 2014
Neutrophils mediate blood-spinal cord barrier disruption in demyelinating neuroinflammatory diseasesBenoit Aubé, Sébastien A Lévesque, Alexandre Paré, et al.
The Journal of Experimental Medicine|May 4, 2016
Myeloid cell transmigration across the CNS vasculature triggers IL-1β-driven neuroinflammation during autoimmune encephalomyelitis in miceSébastien A Lévesque, Alexandre Paré, Benoit Mailhot, et al.
Blood|August 2, 2014
Platelets release mitochondria serving as substrate for bactericidal group IIA-secreted phospholipase A2 to promote inflammationLuc H Boudreau, Anne-Claire Duchez, Nathalie Cloutier, et al.
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