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Alexei V Tepikin

Showing results (51-60 of 70) with videos related to

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Frontiers in Physiology|July 28, 2018
F<sub>1</sub>F<sub>0</sub>-ATP Synthase Inhibitory Factor 1 in the Normal Pancreas and in Pancreatic Ductal Adenocarcinoma: Effects on Bioenergetics, Invasion and ProliferationHelen Tanton, Svetlana Voronina, Anthony Evans, et al.
The Journal of Biological Chemistry|April 8, 2018
Oxidative stress alters mitochondrial bioenergetics and modifies pancreatic cell death independently of cyclophilin D, resulting in an apoptosis-to-necrosis shiftJane A Armstrong, Nicole J Cash, Yulin Ouyang, et al.
Gastroenterology|January 28, 2010
Dynamic changes in cytosolic and mitochondrial ATP levels in pancreatic acinar cellsSvetlana G Voronina, Stephanie L Barrow, Alec W M Simpson, et al.
The Biochemical Journal|November 6, 2014
The role of Ca2+ influx in endocytic vacuole formation in pancreatic acinar cellsSvetlana Voronina, David Collier, Michael Chvanov, et al.
The Journal of Biological Chemistry|June 12, 2009
Calcium elevation in mitochondria is the main Ca2+ requirement for mitochondrial permeability transition pore (mPTP) openingHeidi K Baumgartner, Julia V Gerasimenko, Christopher Thorne, et al.
Gastroenterology|March 1, 2011
Reactive oxygen species induced by bile acid induce apoptosis and protect against necrosis in pancreatic acinar cellsDavid M Booth, John A Murphy, Rajarshi Mukherjee, et al.
Cells|June 11, 2020
Knockout of the Mitochondrial Calcium Uniporter Strongly Suppresses Stimulus-Metabolism Coupling in Pancreatic Acinar Cells but Does Not Reduce Severity of Experimental Acute PancreatitisMichael Chvanov, Svetlana Voronina, Xiaoying Zhang, et al.
Pflugers Archiv : European Journal of Physiology|October 24, 2007
ATP depletion inhibits Ca2+ release, influx and extrusion in pancreatic acinar cells but not pathological Ca2+ responses induced by bileStephanie L Barrow, Svetlana G Voronina, Gabriela da Silva Xavier, et al.
The Biochemical Journal|January 14, 2016
Epithelial-mesenchymal transition, IP3 receptors and ER-PM junctions: translocation of Ca2+ signalling complexes and regulation of migrationEmmanuel Okeke, Tony Parker, Hayley Dingsdale, et al.
Pflugers Archiv : European Journal of Physiology|June 11, 2008
ATP depletion induces translocation of STIM1 to puncta and formation of STIM1-ORAI1 clusters: translocation and re-translocation of STIM1 does not require ATPMichael Chvanov, Ciara M Walsh, Lee P Haynes, et al.
Pageof 7

Showing results (51-60 of 70) with videos related to

Sort By:
Pageof 7
Frontiers in Physiology|July 28, 2018
F<sub>1</sub>F<sub>0</sub>-ATP Synthase Inhibitory Factor 1 in the Normal Pancreas and in Pancreatic Ductal Adenocarcinoma: Effects on Bioenergetics, Invasion and ProliferationHelen Tanton, Svetlana Voronina, Anthony Evans, et al.
The Journal of Biological Chemistry|April 8, 2018
Oxidative stress alters mitochondrial bioenergetics and modifies pancreatic cell death independently of cyclophilin D, resulting in an apoptosis-to-necrosis shiftJane A Armstrong, Nicole J Cash, Yulin Ouyang, et al.
Gastroenterology|January 28, 2010
Dynamic changes in cytosolic and mitochondrial ATP levels in pancreatic acinar cellsSvetlana G Voronina, Stephanie L Barrow, Alec W M Simpson, et al.
The Biochemical Journal|November 6, 2014
The role of Ca2+ influx in endocytic vacuole formation in pancreatic acinar cellsSvetlana Voronina, David Collier, Michael Chvanov, et al.
The Journal of Biological Chemistry|June 12, 2009
Calcium elevation in mitochondria is the main Ca2+ requirement for mitochondrial permeability transition pore (mPTP) openingHeidi K Baumgartner, Julia V Gerasimenko, Christopher Thorne, et al.
Gastroenterology|March 1, 2011
Reactive oxygen species induced by bile acid induce apoptosis and protect against necrosis in pancreatic acinar cellsDavid M Booth, John A Murphy, Rajarshi Mukherjee, et al.
Cells|June 11, 2020
Knockout of the Mitochondrial Calcium Uniporter Strongly Suppresses Stimulus-Metabolism Coupling in Pancreatic Acinar Cells but Does Not Reduce Severity of Experimental Acute PancreatitisMichael Chvanov, Svetlana Voronina, Xiaoying Zhang, et al.
Pflugers Archiv : European Journal of Physiology|October 24, 2007
ATP depletion inhibits Ca2+ release, influx and extrusion in pancreatic acinar cells but not pathological Ca2+ responses induced by bileStephanie L Barrow, Svetlana G Voronina, Gabriela da Silva Xavier, et al.
The Biochemical Journal|January 14, 2016
Epithelial-mesenchymal transition, IP3 receptors and ER-PM junctions: translocation of Ca2+ signalling complexes and regulation of migrationEmmanuel Okeke, Tony Parker, Hayley Dingsdale, et al.
Pflugers Archiv : European Journal of Physiology|June 11, 2008
ATP depletion induces translocation of STIM1 to puncta and formation of STIM1-ORAI1 clusters: translocation and re-translocation of STIM1 does not require ATPMichael Chvanov, Ciara M Walsh, Lee P Haynes, et al.
Pageof 7