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American Journal of Physiology. Heart and Circulatory Physiology|July 23, 2013
Does the combination of hyperkalemia and KATP activation determine excitation rate gradient and electrical failure in the globally ischemic fibrillating heart?Tyson G Taylor, Paul W Venable, Alicja Booth, et al.
American Journal of Physiology. Heart and Circulatory Physiology|March 16, 2019
Conduction in the right and left ventricle is differentially regulated by protein kinases and phosphatases: implications for arrhythmogenesisAlexey V Zaitsev, Natalia S Torres, Keiko M Cawley, et al.
American Journal of Physiology. Heart and Circulatory Physiology|January 2, 2015
Mitochondrial depolarization and asystole in the globally ischemic rabbit heart: coordinated response to interventions affecting energy balancePaul W Venable, Katie J Sciuto, Mark Warren, et al.
Cardiovascular Research|December 29, 2004
Effect of remodelling, stretch and ischaemia on ventricular fibrillation frequency and dynamics in a heart failure modelJavier Moreno, Alexey V Zaitsev, Mark Warren, et al.
American Journal of Physiology. Heart and Circulatory Physiology|June 5, 2007
Three distinct phases of VF during global ischemia in the isolated blood-perfused pig heartJose F Huizar, Mark D Warren, Alexander G Shvedko, et al.
American Journal of Physiology. Heart and Circulatory Physiology|January 29, 2017
Blockade of CaMKII depresses conduction preferentially in the right ventricular outflow tract and promotes ischemic ventricular fibrillation in the rabbit heartMark Warren, Katie J Sciuto, Tyson G Taylor, et al.
Journal of Cardiovascular Electrophysiology|February 18, 2009
Modulation of the sinus rate during ventricular fibrillationStephen L Wasmund, Rakesh K Pai, Roger A Freedman, et al.
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