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Alfred L Goldberg

Showing results (71-80 of 143) with videos related to

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American Journal of Physiology. Endocrinology and Metabolism|April 22, 2004
IGF-I stimulates muscle growth by suppressing protein breakdown and expression of atrophy-related ubiquitin ligases, atrogin-1 and MuRF1Jennifer M Sacheck, Akira Ohtsuka, S Christine McLary, et al.
Molecular Cell|April 1, 2004
Yeast adapt to near-freezing temperatures by STRE/Msn2,4-dependent induction of trehalose synthesis and certain molecular chaperonesOlga Kandror, Nancy Bretschneider, Evgeniy Kreydin, et al.
Biomolecules|June 2, 2021
Mechanisms That Activate 26S Proteasomes and Enhance Protein DegradationAlfred L Goldberg, Hyoung Tae Kim, Donghoon Lee, et al.
The EMBO Journal|May 29, 2002
Properties of the hybrid form of the 26S proteasome containing both 19S and PA28 complexesPaolo Cascio, Matthew Call, Benjamin M Petre, et al.
Cell|February 22, 2011
ATP binds to proteasomal ATPases in pairs with distinct functional effects, implying an ordered reaction cycleDavid M Smith, Hugo Fraga, Christian Reis, et al.
The Journal of Cell Biology|February 26, 2014
Trim32 reduces PI3K-Akt-FoxO signaling in muscle atrophy by promoting plakoglobin-PI3K dissociationShenhav Cohen, Donghoon Lee, Bo Zhai, et al.
The Journal of Biological Chemistry|March 31, 2022
Mammalian Ddi2 is a shuttling factor containing a retroviral protease domain that influences binding of ubiquitylated proteins and proteasomal degradationGalen Andrew Collins, Zhe Sha, Chueh-Ling Kuo, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 20, 2006
Tripeptidyl peptidase II is the major peptidase needed to trim long antigenic precursors, but is not required for most MHC class I antigen presentationIan A York, Nidhi Bhutani, Sophia Zendzian, et al.
Trends in Immunology|February 26, 2014
Re-examining class-I presentation and the DRiP hypothesisKenneth L Rock, Diego J Farfán-Arribas, Jeff D Colbert, et al.
Molecular Cell|December 13, 2005
ATP binding to PAN or the 26S ATPases causes association with the 20S proteasome, gate opening, and translocation of unfolded proteinsDavid M Smith, Galit Kafri, Yifan Cheng, et al.
Pageof 15

Showing results (71-80 of 143) with videos related to

Sort By:
Pageof 15
American Journal of Physiology. Endocrinology and Metabolism|April 22, 2004
IGF-I stimulates muscle growth by suppressing protein breakdown and expression of atrophy-related ubiquitin ligases, atrogin-1 and MuRF1Jennifer M Sacheck, Akira Ohtsuka, S Christine McLary, et al.
Molecular Cell|April 1, 2004
Yeast adapt to near-freezing temperatures by STRE/Msn2,4-dependent induction of trehalose synthesis and certain molecular chaperonesOlga Kandror, Nancy Bretschneider, Evgeniy Kreydin, et al.
Biomolecules|June 2, 2021
Mechanisms That Activate 26S Proteasomes and Enhance Protein DegradationAlfred L Goldberg, Hyoung Tae Kim, Donghoon Lee, et al.
The EMBO Journal|May 29, 2002
Properties of the hybrid form of the 26S proteasome containing both 19S and PA28 complexesPaolo Cascio, Matthew Call, Benjamin M Petre, et al.
Cell|February 22, 2011
ATP binds to proteasomal ATPases in pairs with distinct functional effects, implying an ordered reaction cycleDavid M Smith, Hugo Fraga, Christian Reis, et al.
The Journal of Cell Biology|February 26, 2014
Trim32 reduces PI3K-Akt-FoxO signaling in muscle atrophy by promoting plakoglobin-PI3K dissociationShenhav Cohen, Donghoon Lee, Bo Zhai, et al.
The Journal of Biological Chemistry|March 31, 2022
Mammalian Ddi2 is a shuttling factor containing a retroviral protease domain that influences binding of ubiquitylated proteins and proteasomal degradationGalen Andrew Collins, Zhe Sha, Chueh-Ling Kuo, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 20, 2006
Tripeptidyl peptidase II is the major peptidase needed to trim long antigenic precursors, but is not required for most MHC class I antigen presentationIan A York, Nidhi Bhutani, Sophia Zendzian, et al.
Trends in Immunology|February 26, 2014
Re-examining class-I presentation and the DRiP hypothesisKenneth L Rock, Diego J Farfán-Arribas, Jeff D Colbert, et al.
Molecular Cell|December 13, 2005
ATP binding to PAN or the 26S ATPases causes association with the 20S proteasome, gate opening, and translocation of unfolded proteinsDavid M Smith, Galit Kafri, Yifan Cheng, et al.
Pageof 15