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American Journal of Physiology. Renal Physiology|September 17, 2011
Acute unilateral ischemic renal injury induces progressive renal inflammation, lipid accumulation, histone modification, and "end-stage" kidney diseaseRichard A Zager, Ali C M Johnson, Kirsten Becker
American Journal of Physiology. Renal Physiology|May 13, 2016
An evaluation of the antioxidant protein α1-microglobulin as a renal tubular cytoprotectantRichard A Zager, Ali C M Johnson, Kirsten Frostad
Journal of the American Society of Nephrology : JASN|March 24, 2012
Plasma and urinary heme oxygenase-1 in AKIRichard A Zager, Ali C M Johnson, Kirsten Becker
Kidney International|May 11, 2005
Triglyceride accumulation in injured renal tubular cells: alterations in both synthetic and catabolic pathwaysAli C M Johnson, Andreas Stahl, Richard A Zager
American Journal of Physiology. Renal Physiology|August 19, 2007
Gentamicin suppresses endotoxin-driven TNF-alpha production in human and mouse proximal tubule cellsRichard A Zager, Ali C M Johnson, Adam Geballe
American Journal of Physiology. Renal Physiology|May 11, 2012
Proximal tubule haptoglobin gene activation is an integral component of the acute kidney injury "stress response"Richard A Zager, Anitha Vijayan, Ali C M Johnson
American Journal of Physiology. Renal Physiology|May 28, 2010
Parenteral iron formulations differentially affect MCP-1, HO-1, and NGAL gene expression and renal responses to injuryAli C M Johnson, Kirsten Becker, Richard A Zager
Journal of the American Society of Nephrology : JASN|January 4, 2014
Renal cortical pyruvate depletion during AKIRichard A Zager, Ali C M Johnson, Kirsten Becker
American Journal of Physiology. Renal Physiology|September 21, 2012
Renal cortical hemopexin accumulation in response to acute kidney injuryRichard A Zager, Ali C M Johnson, Kirsten Becker
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