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Arteriosclerosis, Thrombosis, and Vascular Biology|June 13, 2015
Plasma apolipoprotein C-III levels, triglycerides, and coronary artery calcification in type 2 diabeticsArman Qamar, Sumeet A Khetarpal, Amit V Khera, et al.
European Journal of Human Genetics : EJHG|September 10, 2015
Phenotypic extremes in rare variant study designsGina M Peloso, Daniel J Rader, Stacey Gabriel, et al.
Journal of Lipid Research|May 29, 2002
Characterization of the lipolytic activity of endothelial lipaseMary G McCoy, Gwo-Shing Sun, Dawn Marchadier, et al.
Circulation|August 6, 2003
Overexpression of apolipoprotein A-I promotes reverse transport of cholesterol from macrophages to feces in vivoYuZhen Zhang, Ilaria Zanotti, Muredach P Reilly, et al.
Circulation|February 16, 2005
Resistin is an inflammatory marker of atherosclerosis in humansMuredach P Reilly, Michael Lehrke, Megan L Wolfe, et al.
The Journal of Heart and Lung Transplantation : the Official Publication of the International Society for Heart Transplantation|August 8, 2016
Cholesterol efflux capacity of high-density lipoprotein correlates with survival and allograft vasculopathy in cardiac transplant recipientsAli Javaheri, Maria Molina, Payman Zamani, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|June 2, 2012
Genetic ablation of Adamts13 gene dramatically accelerates the formation of early atherosclerosis in a murine modelSheng-Yu Jin, Junichiro Tohyama, Robert C Bauer, et al.
The Journal of Clinical Investigation|October 4, 2005
Hepatic expression of scavenger receptor class B type I (SR-BI) is a positive regulator of macrophage reverse cholesterol transport in vivoYuZhen Zhang, Jaqueline R Da Silva, Muredach Reilly, et al.
Mammalian Genome : Official Journal of the International Mammalian Genome Society|June 1, 2013
A coding variant in SR-BI (I179N) significantly increases atherosclerosis in miceAntonino Picataggi, Geoffrey F Lim, Anthony P Kent, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|July 16, 2002
Human secretory phospholipase A2 mediates decreased plasma levels of HDL cholesterol and apoA-I in response to inflammation in human apoA-I transgenic miceUwe J F Tietge, Cyrille Maugeais, Sissel Lund-Katz, et al.
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