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Pediatric Research|December 7, 2017
Improving clinical diagnosis in SHOX deficiency: the importance of growth velocityGiulia Genoni, Alice Monzani, Matteo Castagno, et al.
Children (Basel, Switzerland)|November 27, 2021
Changing Admission Patterns in Pediatric Emergency Departments during the COVID-19 Pandemic in Italy Were Due to Reductions in Inappropriate AccessesIvana Rabbone, Francesco Tagliaferri, Elena Carboni, et al.
Plos One|March 6, 2018
High-normal estimated glomerular filtration rate and hyperuricemia positively correlate with metabolic impairment in pediatric obese patientsRoberta Ricotti, Giulia Genoni, Enza Giglione, et al.
Molecular Genetics & Genomic Medicine|November 23, 2021
Copy number variations residing outside the SHOX enhancer region are involved in Short Stature and Léri-Weill dyschondrosteosisAntonella Fanelli, Silvia Vannelli, Deepak Babu, et al.
American Heart Journal|September 15, 2019
Small-for-gestational-age birth is linked to cardiovascular dysfunction in early childhoodMatteo Castagno, Veronica Menegon, Alice Monzani, et al.
The Journal of Pediatrics|January 23, 2016
Influence of Ultraviolet Radiation on the Association between 25-Hydroxy Vitamin D Levels and Cardiovascular Risk Factors in ObesityFlavia Prodam, Sara Zanetta, Roberta Ricotti, et al.
Diabetes, Obesity & Metabolism|January 9, 2025
Efficacy of myo-inositol and zinc on insulin resistance in a paediatric population with obesityValentina Antoniotti, Cristina Partenope, Arianna Solito, et al.
International Journal of Cardiology|September 23, 2017
Insulin resistance, serum uric acid and metabolic syndrome are linked to cardiovascular dysfunction in pediatric obesityGiulia Genoni, Veronica Menegon, Gioel Gabrio Secco, et al.
Biology Direct|April 1, 2025
The CXCR3/PLC/IP3-IP3R axis is responsible for the ignition of UPR in intestinal epithelial cells exposed to gliadin peptide, during the onset of celiac diseaseRomina Monzani, Mara Gagliardi, Valentina Saverio, et al.
Biology Direct|April 17, 2025
Gliadin-dependent UPR induction directly triggers the expression of TG2 and pro-inflammatory cytokines, dysregulates intestinal permeability, and reduces CFTR expression in intestinal epithelial cells of celiac disease patientsRomina Monzani, Mara Gagliardi, Valentina Saverio, et al.
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