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Alicia Mattiazzi

Showing results (21-30 of 52) with videos related to

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The Journal of Physiology|February 5, 2025
CaMKII at the crossroads: calcium dysregulation, and post-translational modifications driving cell deathAlicia Mattiazzi, Carolina Jaquenod De Giusti, Carlos A Valverde
Frontiers in Physiology|February 11, 2020
Unbalance Between Sarcoplasmic Reticulum Ca<sup>2 +</sup> Uptake and Release: A First Step Toward Ca<sup>2 +</sup> Triggered Arrhythmias and Cardiac DamageMarilén Federico, Carlos A Valverde, Alicia Mattiazzi, et al.
Molecular and Cellular Biochemistry|October 28, 2003
Phospholamban phosphorylation in ischemia-reperfused heart. Effect of pacing during ischemia and response to a beta-adrenergic challengeCecilia Mundiña-Weilenmann, Matilde Said, Leticia Vittone, et al.
Cardiovascular Research|October 18, 2005
Role of phospholamban phosphorylation on Thr17 in cardiac physiological and pathological conditionsAlicia Mattiazzi, Cecilia Mundiña-Weilenmann, Chu Guoxiang, et al.
Journal of Molecular and Cellular Cardiology|January 29, 2002
Time course and mechanisms of phosphorylation of phospholamban residues in ischemia-reperfused rat hearts. Dissociation of phospholamban phosphorylation pathwaysLeticia Vittone, Cecilia Mundiña-Weilenmann, Matilde Said, et al.
The Journal of Experimental Biology|February 17, 2006
Contractile recovery from acidosis in toad ventricle is independent of intracellular pH and relies upon Ca2+ influxMargarita A Salas, Martín G Vila-Petroff, Roque A Venosa, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 2, 2005
Angiotensin II-induced negative inotropy in rat ventricular myocytes: role of reactive oxygen species and p38 MAPKJulieta Palomeque, Luciana Sapia, Roger J Hajjar, et al.
Journal of Molecular and Cellular Cardiology|December 3, 2014
Ca2+ Sparks and Ca2+ waves are the subcellular events underlying Ca2+ overload during ischemia and reperfusion in perfused intact heartsAlicia Mattiazzi, Mariana Argenziano, Yuriana Aguilar-Sanchez, et al.
Cardiovascular Research|March 17, 2005
Role of phosphorylation of Thr(17) residue of phospholamban in mechanical recovery during hypercapnic acidosisCecilia Mundiña-Weilenmann, Paola Ferrero, Matilde Said, et al.
Scientific Reports|August 22, 2018
SERCA is critical to control the Bowditch effect in the heartDarío Balcazar, Victoria Regge, Manuela Santalla, et al.
Pageof 6

Showing results (21-30 of 52) with videos related to

Sort By:
Pageof 6
The Journal of Physiology|February 5, 2025
CaMKII at the crossroads: calcium dysregulation, and post-translational modifications driving cell deathAlicia Mattiazzi, Carolina Jaquenod De Giusti, Carlos A Valverde
Frontiers in Physiology|February 11, 2020
Unbalance Between Sarcoplasmic Reticulum Ca<sup>2 +</sup> Uptake and Release: A First Step Toward Ca<sup>2 +</sup> Triggered Arrhythmias and Cardiac DamageMarilén Federico, Carlos A Valverde, Alicia Mattiazzi, et al.
Molecular and Cellular Biochemistry|October 28, 2003
Phospholamban phosphorylation in ischemia-reperfused heart. Effect of pacing during ischemia and response to a beta-adrenergic challengeCecilia Mundiña-Weilenmann, Matilde Said, Leticia Vittone, et al.
Cardiovascular Research|October 18, 2005
Role of phospholamban phosphorylation on Thr17 in cardiac physiological and pathological conditionsAlicia Mattiazzi, Cecilia Mundiña-Weilenmann, Chu Guoxiang, et al.
Journal of Molecular and Cellular Cardiology|January 29, 2002
Time course and mechanisms of phosphorylation of phospholamban residues in ischemia-reperfused rat hearts. Dissociation of phospholamban phosphorylation pathwaysLeticia Vittone, Cecilia Mundiña-Weilenmann, Matilde Said, et al.
The Journal of Experimental Biology|February 17, 2006
Contractile recovery from acidosis in toad ventricle is independent of intracellular pH and relies upon Ca2+ influxMargarita A Salas, Martín G Vila-Petroff, Roque A Venosa, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 2, 2005
Angiotensin II-induced negative inotropy in rat ventricular myocytes: role of reactive oxygen species and p38 MAPKJulieta Palomeque, Luciana Sapia, Roger J Hajjar, et al.
Journal of Molecular and Cellular Cardiology|December 3, 2014
Ca2+ Sparks and Ca2+ waves are the subcellular events underlying Ca2+ overload during ischemia and reperfusion in perfused intact heartsAlicia Mattiazzi, Mariana Argenziano, Yuriana Aguilar-Sanchez, et al.
Cardiovascular Research|March 17, 2005
Role of phosphorylation of Thr(17) residue of phospholamban in mechanical recovery during hypercapnic acidosisCecilia Mundiña-Weilenmann, Paola Ferrero, Matilde Said, et al.
Scientific Reports|August 22, 2018
SERCA is critical to control the Bowditch effect in the heartDarío Balcazar, Victoria Regge, Manuela Santalla, et al.
Pageof 6