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European Journal of Immunology|May 13, 2026
Integrating Genetics and Environment to Find Causal Mechanisms for Multiple SclerosisAlicia Munoz Leon, Matthew R LincolnNeurology|March 20, 2023
Pearls & Oy-sters: Homozygous Complement Factor I Deficiency Presenting as Fulminant Relapsing Complement-Mediated CNS VasculitisElle Levit, Janice Leon, Matthew R LincolnEuropean Journal of Clinical Investigation|March 23, 2011
Of mice and men: experimental autoimmune encephalitis and multiple sclerosisAdam E Handel, Matthew R Lincoln, Sreeram V RamagopalanCurrent Opinion in Immunology|September 25, 2020
Epigenetic fine-mapping: identification of causal mechanisms for autoimmunityMatthew R Lincoln, Pierre-Paul Axisa, David A HaflerHuman Molecular Genetics|December 23, 2008
Epigenetics in multiple sclerosis susceptibility: difference in transgenerational risk localizes to the major histocompatibility complexMichael J Chao, Sreeram V Ramagopalan, Blanca M Herrera, et al.Human Molecular Genetics|June 3, 2005
Complex interactions among MHC haplotypes in multiple sclerosis: susceptibility and resistanceDavid A Dyment, Blanca M Herrera, M Zameel Cader, et al.Human Molecular Genetics|July 17, 2010
Parent-of-origin effects at the major histocompatibility complex in multiple sclerosisMichael J Chao, Blanca M Herrera, Sreeram V Ramagopalan, et al.Nature Immunology|October 31, 2018
Activated β-catenin in Foxp3+ regulatory T cells links inflammatory environments to autoimmunityTomokazu Sumida, Matthew R Lincoln, Chinonso M Ukeje, et al.The American Journal of Clinical Nutrition|August 12, 2008
Evidence for genetic regulation of vitamin D status in twins with multiple sclerosisSarah-Michelle Orton, Andrew P Morris, Blanca M Herrera, et al.Nature Immunology|June 9, 2019
Author Correction: Activated β-catenin in Foxp3+ regulatory T cells links inflammatory environments to autoimmunityTomokazu Sumida, Matthew R Lincoln, Chinonso M Ukeje, et al.Pageof 4