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The Journal of Biological Chemistry|January 21, 2012
Neurodegeneration induced by clustering of sialylated glycosylphosphatidylinositols of prion proteinsClive Bate, Alun WilliamsThe Journal of Laryngology and Otology|May 1, 2004
Early experience with the Mills sleeve prosthesis for reconstruction of the incus long processRobert Mills, Alun WilliamsNeuropharmacology|September 10, 2011
Ethanol protects cultured neurons against amyloid-β and α-synuclein-induced synapse damageClive Bate, Alun WilliamsPrion|July 9, 2011
The cellular prion protein with a monoacylated glycosylphosphatidylinositol anchor modifies cell membranes, inhibits cell signaling and reduces prion formationClive Bate, Alun WilliamsPrion|August 17, 2012
Clustering of sialylated glycosylphosphatidylinositol anchors mediates PrP-induced activation of cytoplasmic phospholipase A 2 and synapse damageClive Bate, Alun WilliamsThe Journal of General Virology|November 24, 2004
Role of glycosylphosphatidylinositols in the activation of phospholipase A2 and the neurotoxicity of prionsClive Bate, Alun WilliamsNeurobiology of Disease|December 23, 2017
Monomeric amyloid-β reduced amyloid-β oligomer-induced synapse damage in neuronal culturesClive Bate, Alun WilliamsBiomolecules|March 12, 2015
α-Synuclein-induced synapse damage in cultured neurons is mediated by cholesterol-sensitive activation of cytoplasmic phospholipase A2Clive Bate, Alun WilliamsJournal of Alzheimer'S Disease : JAD|July 17, 2010
Amyloid-β(1-40) inhibits amyloid-β(1-42) induced activation of cytoplasmic phospholipase A2 and synapse degenerationClive Bate, Alun WilliamsThe Journal of Biological Chemistry|January 8, 2011
Monoacylated cellular prion protein modifies cell membranes, inhibits cell signaling, and reduces prion formationClive Bate, Alun WilliamsPageof 9