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Iscience|September 15, 2021
Encephalopathy-causing mutations in Gβ1 (GNB1) alter regulation of neuronal GIRK channelsHaritha P Reddy, Daniel Yakubovich, Tal Keren-Raifman, et al.Journal of Cell Science|February 24, 2012
Molecular interactions of the physiological anti-hypertensive peptide catestatin with the neuronal nicotinic acetylcholine receptorBhavani S Sahu, Jagan Mohan, Jagan M Obbineni, et al.The Journal of Biological Chemistry|October 30, 2012
Functional genetic variants of the catecholamine-release-inhibitory peptide catestatin in an Indian population: allele-specific effects on metabolic traitsBhavani S Sahu, Jagan M Obbineni, Giriraj Sahu, et al.Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|October 15, 2022
Intoxicating effects of alcohol depend on acid-sensing ion channelsGail I S Harmata, Aubrey C Chan, Madison J Merfeld, et al.Frontiers in Cellular Neuroscience|June 5, 2023
Epilepsy in a mouse model of GNB1 encephalopathy arises from altered potassium (GIRK) channel signaling and is alleviated by a GIRK inhibitorSophie Colombo, Haritha P Reddy, Sabrina Petri, et al.Pageof 2