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Amanda K Huber

Showing results (1-10 of 43) with videos related to

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Frontiers in Oncology|May 15, 2015
Is the concept of central nervous system immune privilege irrelevant in the setting of acute infection?Amanda K Huber, David N Irani
Advances in Neuroimmune Biology|February 9, 2016
Targeting CXCL13 During NeuroinflammationAmanda K Huber, David N Irani
Frontiers in Oncology|November 29, 2014
Immune responses to non-tumor antigens in the central nervous systemAmanda K Huber, Patrick C Duncker, David N Irani
Cytokine|February 19, 2015
The conundrum of interferon-β non-responsiveness in relapsing-remitting multiple sclerosisAmanda K Huber, Patrick C Duncker, David N Irani
Journal of Neuroinflammation|August 27, 2016
Type-1 angiotensin receptor signaling in central nervous system myeloid cells is pathogenic during fatal alphavirus encephalitis in micePennelope K Blakely, Amanda K Huber, David N Irani
Thyroid : Official Journal of the American Thyroid Association|December 25, 2012
Hepatitis C virus infection of a thyroid cell line: implications for pathogenesis of hepatitis C virus and thyroiditisJason T Blackard, Ling Kong, Amanda K Huber, et al.
Clinical Immunology (Orlando, Fla.)|September 25, 2016
An emerging role for eotaxins in neurodegenerative diseaseAmanda K Huber, David A Giles, Benjamin M Segal, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 31, 2017
GM-CSF Promotes Chronic Disability in Experimental Autoimmune Encephalomyelitis by Altering the Composition of Central Nervous System-Infiltrating Cells, but Is Dispensable for Disease InductionPatrick C Duncker, Joshua S Stoolman, Amanda K Huber, et al.
Journal of Immunology (Baltimore, Md. : 1950)|June 15, 2014
Site-specific chemokine expression regulates central nervous system inflammation and determines clinical phenotype in autoimmune encephalomyelitisJoshua S Stoolman, Patrick C Duncker, Amanda K Huber, et al.
The Journal of Clinical Endocrinology and Metabolism|December 13, 2007
Interleukin (IL)-23 receptor is a major susceptibility gene for Graves' ophthalmopathy: the IL-23/T-helper 17 axis extends to thyroid autoimmunityAmanda K Huber, Eric M Jacobson, Krystian Jazdzewski, et al.
Pageof 5

Showing results (1-10 of 43) with videos related to

Sort By:
Pageof 5
Frontiers in Oncology|May 15, 2015
Is the concept of central nervous system immune privilege irrelevant in the setting of acute infection?Amanda K Huber, David N Irani
Advances in Neuroimmune Biology|February 9, 2016
Targeting CXCL13 During NeuroinflammationAmanda K Huber, David N Irani
Frontiers in Oncology|November 29, 2014
Immune responses to non-tumor antigens in the central nervous systemAmanda K Huber, Patrick C Duncker, David N Irani
Cytokine|February 19, 2015
The conundrum of interferon-β non-responsiveness in relapsing-remitting multiple sclerosisAmanda K Huber, Patrick C Duncker, David N Irani
Journal of Neuroinflammation|August 27, 2016
Type-1 angiotensin receptor signaling in central nervous system myeloid cells is pathogenic during fatal alphavirus encephalitis in micePennelope K Blakely, Amanda K Huber, David N Irani
Thyroid : Official Journal of the American Thyroid Association|December 25, 2012
Hepatitis C virus infection of a thyroid cell line: implications for pathogenesis of hepatitis C virus and thyroiditisJason T Blackard, Ling Kong, Amanda K Huber, et al.
Clinical Immunology (Orlando, Fla.)|September 25, 2016
An emerging role for eotaxins in neurodegenerative diseaseAmanda K Huber, David A Giles, Benjamin M Segal, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 31, 2017
GM-CSF Promotes Chronic Disability in Experimental Autoimmune Encephalomyelitis by Altering the Composition of Central Nervous System-Infiltrating Cells, but Is Dispensable for Disease InductionPatrick C Duncker, Joshua S Stoolman, Amanda K Huber, et al.
Journal of Immunology (Baltimore, Md. : 1950)|June 15, 2014
Site-specific chemokine expression regulates central nervous system inflammation and determines clinical phenotype in autoimmune encephalomyelitisJoshua S Stoolman, Patrick C Duncker, Amanda K Huber, et al.
The Journal of Clinical Endocrinology and Metabolism|December 13, 2007
Interleukin (IL)-23 receptor is a major susceptibility gene for Graves' ophthalmopathy: the IL-23/T-helper 17 axis extends to thyroid autoimmunityAmanda K Huber, Eric M Jacobson, Krystian Jazdzewski, et al.
Pageof 5