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The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 16, 2018
Modulation of Receptor Protein Tyrosine Phosphatase Sigma Increases Chondroitin Sulfate Proteoglycan Degradation through Cathepsin B Secretion to Enhance Axon OutgrowthAmanda Phuong Tran, Sapna Sundar, Meigen Yu, et al.Microsurgery|January 29, 2021
A meta-analysis of functional outcomes in rat sciatic nerve injury modelsAnthony DeLeonibus, Majid Rezaei, Vahe Fahradyan, et al.Nature|July 15, 2011
Functional regeneration of respiratory pathways after spinal cord injuryWarren J Alilain, Kevin P Horn, Hongmei Hu, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 12, 2002
The critical role of basement membrane-independent laminin gamma 1 chain during axon regeneration in the CNSBarbara Grimpe, Sucai Dong, Catherine Doller, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 15, 2010
Serotonergic neurons migrate radially through the neuroepithelium by dynamin-mediated somal translocationAlicia L Hawthorne, Christi J Wylie, Lynn T Landmesser, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 14, 2006
Combining an autologous peripheral nervous system "bridge" and matrix modification by chondroitinase allows robust, functional regeneration beyond a hemisection lesion of the adult rat spinal cordJohn D Houle, Veronica J Tom, Debra Mayes, et al.Proceedings of the National Academy of Sciences of the United States of America|November 15, 2017
Phasic inhibition as a mechanism for generation of rapid respiratory rhythmsJared M Cregg, Kevin A Chu, Thomas E Dick, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 23, 2004
Studies on the development and behavior of the dystrophic growth cone, the hallmark of regeneration failure, in an in vitro model of the glial scar and after spinal cord injuryVeronica J Tom, Michael P Steinmetz, Jared H Miller, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 19, 2008
Another barrier to regeneration in the CNS: activated macrophages induce extensive retraction of dystrophic axons through direct physical interactionsKevin P Horn, Sarah A Busch, Alicia L Hawthorne, et al.Experimental Neurology|November 15, 2011
Oncomodulin affords limited regeneration to injured sensory axons in vitro and in vivoRan Harel, Christopher A Iannotti, Daniel Hoh, et al.Pageof 8