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Amanda Penco

Showing results (11-20 of 21) with videos related to

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Human Molecular Genetics|June 22, 2017
Epigallocatechin-3-gallate and related phenol compounds redirect the amyloidogenic aggregation pathway of ataxin-3 towards non-toxic aggregates and prevent toxicity in neural cells and Caenorhabditis elegans animal modelCristina Visentin, Francesca Pellistri, Antonino Natalello, et al.
Biological Chemistry|January 27, 2016
Effect of molecular chaperones on aberrant protein oligomers in vitro: super-versus sub-stoichiometric chaperone concentrationsSara Cappelli, Amanda Penco, Benedetta Mannini, et al.
Human Molecular Genetics|July 18, 2014
Epigallocatechin-3-gallate and tetracycline differently affect ataxin-3 fibrillogenesis and reduce toxicity in spinocerebellar ataxia type 3 modelMarcella Bonanomi, Antonino Natalello, Cristina Visentin, et al.
Biochimica Et Biophysica Acta|September 17, 2013
Different ataxin-3 amyloid aggregates induce intracellular Ca(2+) deregulation by different mechanisms in cerebellar granule cellsFrancesca Pellistri, Monica Bucciantini, Gaetano Invernizzi, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 16, 2015
Nucleophosmin contains amyloidogenic regions that are able to form toxic aggregates under physiological conditionsConcetta Di Natale, Pasqualina Liana Scognamiglio, Roberta Cascella, et al.
Nucleic Acids Research|May 31, 2011
The biological and structural characterization of Mycobacterium tuberculosis UvrA provides novel insights into its mechanism of actionFranca Rossi, Jasbeer Singh Khanduja, Alessio Bortoluzzi, et al.
Biochimica Et Biophysica Acta|October 31, 2015
Protein conformational perturbations in hereditary amyloidosis: Differential impact of single point mutations in ApoAI amyloidogenic variantsRita Del Giudice, Angela Arciello, Francesco Itri, et al.
Biophysical Journal|November 3, 2016
Biochemical and Electrophysiological Modification of Amyloid Transthyretin on CardiomyocytesLaura Sartiani, Monica Bucciantini, Valentina Spinelli, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 18, 2012
Molecular mechanisms used by chaperones to reduce the toxicity of aberrant protein oligomersBenedetta Mannini, Roberta Cascella, Mariagioia Zampagni, et al.
The Journal of Biological Chemistry|December 16, 2014
The H50Q mutation induces a 10-fold decrease in the solubility of α-synucleinRiccardo Porcari, Christos Proukakis, Christopher A Waudby, et al.
Pageof 3

Showing results (11-20 of 21) with videos related to

Sort By:
Pageof 3
Human Molecular Genetics|June 22, 2017
Epigallocatechin-3-gallate and related phenol compounds redirect the amyloidogenic aggregation pathway of ataxin-3 towards non-toxic aggregates and prevent toxicity in neural cells and Caenorhabditis elegans animal modelCristina Visentin, Francesca Pellistri, Antonino Natalello, et al.
Biological Chemistry|January 27, 2016
Effect of molecular chaperones on aberrant protein oligomers in vitro: super-versus sub-stoichiometric chaperone concentrationsSara Cappelli, Amanda Penco, Benedetta Mannini, et al.
Human Molecular Genetics|July 18, 2014
Epigallocatechin-3-gallate and tetracycline differently affect ataxin-3 fibrillogenesis and reduce toxicity in spinocerebellar ataxia type 3 modelMarcella Bonanomi, Antonino Natalello, Cristina Visentin, et al.
Biochimica Et Biophysica Acta|September 17, 2013
Different ataxin-3 amyloid aggregates induce intracellular Ca(2+) deregulation by different mechanisms in cerebellar granule cellsFrancesca Pellistri, Monica Bucciantini, Gaetano Invernizzi, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 16, 2015
Nucleophosmin contains amyloidogenic regions that are able to form toxic aggregates under physiological conditionsConcetta Di Natale, Pasqualina Liana Scognamiglio, Roberta Cascella, et al.
Nucleic Acids Research|May 31, 2011
The biological and structural characterization of Mycobacterium tuberculosis UvrA provides novel insights into its mechanism of actionFranca Rossi, Jasbeer Singh Khanduja, Alessio Bortoluzzi, et al.
Biochimica Et Biophysica Acta|October 31, 2015
Protein conformational perturbations in hereditary amyloidosis: Differential impact of single point mutations in ApoAI amyloidogenic variantsRita Del Giudice, Angela Arciello, Francesco Itri, et al.
Biophysical Journal|November 3, 2016
Biochemical and Electrophysiological Modification of Amyloid Transthyretin on CardiomyocytesLaura Sartiani, Monica Bucciantini, Valentina Spinelli, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 18, 2012
Molecular mechanisms used by chaperones to reduce the toxicity of aberrant protein oligomersBenedetta Mannini, Roberta Cascella, Mariagioia Zampagni, et al.
The Journal of Biological Chemistry|December 16, 2014
The H50Q mutation induces a 10-fold decrease in the solubility of α-synucleinRiccardo Porcari, Christos Proukakis, Christopher A Waudby, et al.
Pageof 3