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Cancer Letters|June 24, 2025
A novel mismatch repair deficient lung adenocarcinoma model for immunotherapy researchIvan Zadra, Etna Abad, Anastasia Krasko, et al.Journal of Experimental & Clinical Cancer Research : CR|April 29, 2024
The TP53-activated E3 ligase RNF144B is a tumour suppressor that prevents genomic instabilityEtna Abad, Jérémy Sandoz, Gerard Romero, et al.Ophthalmic Plastic and Reconstructive Surgery|November 21, 2023
Evaluating ChatGPT on Orbital and Oculofacial Disorders: Accuracy and Readability InsightsMichael Balas, Ana Janic, Patrick Daigle, et al.Cell Death & Disease|October 21, 2020
Consequences of Zmat3 loss in c-MYC- and mutant KRAS-driven tumorigenesisSarah A Best, Cassandra J Vandenberg, Etna Abad, et al.Cell Death and Differentiation|December 19, 2023
Combined absence of TRP53 target genes ZMAT3, PUMA and p21 cause a high incidence of cancer in miceMargs S Brennan, Kerstin Brinkmann, Gerard Romero Sola, et al.Biorxiv : the Preprint Server for Biology|July 17, 2026
Nuclear adenosine metabolism defines a metabolic vulnerability unmasked by TP53 lossJulia Urgel-Solas, Anna Benito-Ferrer, Laura Ortet, et al.Genes & Development|October 28, 2018
Mutant TRP53 exerts a target gene-selective dominant-negative effect to drive tumor developmentBrandon J Aubrey, Ana Janic, Yunshun Chen, et al.Molecular Systems Biology|June 1, 2023
A metabolic map of the DNA damage response identifies PRDX1 in the control of nuclear ROS scavenging and aspartate availabilityAmandine Moretton, Savvas Kourtis, Antoni Gañez Zapater, et al.Science Advances|May 8, 2026
PARP2 deficiency impairs pancreatic cancer progression by promoting genomic instability and antitumor immunityNeus Martínez-Bosch, Noemí Manero-Rupérez, Núria Vázquez-Bellón, et al.Nature Medicine|June 13, 2018
DNA repair processes are critical mediators of p53-dependent tumor suppressionAna Janic, Liz J Valente, Matthew J Wakefield, et al.Pageof 3