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The Lancet. Neurology|February 2, 2015
C9orf72 expansions in frontotemporal dementia and amyotrophic lateral sclerosisJonathan D Rohrer, Adrian M Isaacs, Sarah Mizielinska, et al.Proceedings of the National Academy of Sciences of the United States of America|June 11, 2026
Strain-specific propagation of variant Creutzfeldt-Jakob disease prions in humanized neural cellsMelissa L D Rayner, Parineeta Arora, Jacqueline M Linehan, et al.Acta Neuropathologica|April 22, 2026
Modification of early behavioural, physiological and neuropathological endpoints by syntaxin-6 knockout in a humanised P301S transgenic model of tauopathyElizabeth Hill, Jacqueline Linehan, Michael Farmer, et al.Neuroimage|August 15, 2009
Progressive logopenic/phonological aphasia: erosion of the language networkJonathan D Rohrer, Gerard R Ridgway, Sebastian J Crutch, et al.Journal of Neurology, Neurosurgery, and Psychiatry|February 14, 2014
Altered body schema processing in frontotemporal dementia with C9ORF72 mutationsLaura E Downey, Phillip D Fletcher, Hannah L Golden, et al.Alzheimer'S Research & Therapy|September 26, 2012
Longitudinal neuroimaging and neuropsychological profiles of frontotemporal dementia with C9ORF72 expansionsColin J Mahoney, Laura E Downey, Gerard R Ridgway, et al.Neurobiology of Aging|June 5, 2018
Evaluating the causality of novel sequence variants in the prion protein gene by exampleTze How Mok, Carolin Koriath, Zane Jaunmuktane, et al.The Lancet. Neurology|December 17, 2008
Genetic risk factors for variant Creutzfeldt-Jakob disease: a genome-wide association studySimon Mead, Mark Poulter, James Uphill, et al.Plos Pathogens|February 20, 2025
Isolation of a novel human prion strain from a PRNP codon 129 heterozygous vCJD patientFuquan Zhang, Susan Joiner, Jacqueline M Linehan, et al.Neurology|December 24, 2013
C9orf72 expansions are the most common genetic cause of Huntington disease phenocopiesDavina J Hensman Moss, Mark Poulter, Jon Beck, et al.Pageof 22