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Biorxiv : the Preprint Server for Biology|February 24, 2025
Calcium channel-coupled transcription factors facilitate direct nuclear signalingEshaan R Rao, Tyler Thaxton, Eric Gama, et al.
Frontiers in Digital Health|September 19, 2025
Clinic vs. daily life gait characteristics in patients with spinocerebellar ataxiaVrutangkumar V Shah, Daniel Muzyka, Adam Jagodinsky, et al.
Plos One|April 12, 2011
Frequency of KCNC3 DNA variants as causes of spinocerebellar ataxia 13 (SCA13)Karla P Figueroa, Michael F Waters, Vartan Garibyan, et al.
Neuron|March 30, 2019
α1ACT Is Essential for Survival and Early Cerebellar Programming in a Critical Neonatal WindowXiaofei Du, Cenfu Wei, Daniel Parviz Hejazi Pastor, et al.
Experimental Neurology|December 3, 2014
Fluoxetine is neuroprotective in slow-channel congenital myasthenic syndromeHaipeng Zhu, Gary E Grajales-Reyes, Vivianette Alicea-Vázquez, et al.
Frontiers in Neurology|April 1, 2025
Gait characteristics in people with Friedreich ataxia: daily life versus clinic measuresHannah L Casey, Vrutangkumar V Shah, Daniel Muzyka, et al.
Movement Disorders Clinical Practice|September 21, 2019
Sensitivity of Volumetric Magnetic Resonance Imaging and Magnetic Resonance Spectroscopy to Progression of Spinocerebellar Ataxia Type 1Dinesh K Deelchand, James M Joers, Adarsh Ravishankar, et al.
American Journal of Medical Genetics|July 13, 2002
Childhood-onset ataxia: testing for large CAG-repeats in SCA2 and SCA7Rong Mao, Arthur S Aylsworth, Nicholas Potter, et al.
Molecular and Cellular Neurosciences|April 21, 2006
Novel beta subunit mutation causes a slow-channel syndrome by enhancing activation and decreasing the rate of agonist dissociationManuel F Navedo, José A Lasalde-Dominicci, Carlos A Báez-Pagán, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 22, 2013
Stable respiratory activity requires both P/Q-type and N-type voltage-gated calcium channelsHenner Koch, Sebastien Zanella, Gina E Elsen, et al.
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