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Anant Chopra

Showing results (11-20 of 25) with videos related to

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Developmental Cell|January 10, 2018
Force Generation via β-Cardiac Myosin, Titin, and α-Actinin Drives Cardiac Sarcomere Assembly from Cell-Matrix AdhesionsAnant Chopra, Matthew L Kutys, Kehan Zhang, et al.
Nature|May 31, 2023
Transfer learning enables predictions in network biologyChristina V Theodoris, Ling Xiao, Anant Chopra, et al.
Microsystems & Nanoengineering|September 27, 2021
Probing the subcellular nanostructure of engineered human cardiomyocytes in 3D tissueJosh Javor, Jourdan K Ewoldt, Paige E Cloonan, et al.
Circulation Research|August 18, 2021
Filamin C Cardiomyopathy Variants Cause Protein and Lysosome AccumulationRadhika Agarwal, Joao A Paulo, Christopher N Toepfer, et al.
Circulation. Arrhythmia and Electrophysiology|December 16, 2014
Phospholamban as a crucial determinant of the inotropic response of human pluripotent stem cell-derived ventricular cardiomyocytes and engineered 3-dimensional tissue constructsGaopeng Chen, Sen Li, Ioannis Karakikes, et al.
APL Bioengineering|February 16, 2026
Cell shape and maturation impacts α-actinin-2 tension in iPSC-derived cardiomyocytesPalash K Dutta, Joshua M Toth, Subramanian Sundaram, et al.
Circulation Research|February 1, 2019
SarcTrackChristopher N Toepfer, Arun Sharma, Marcelo Cicconet, et al.
Circulation. Cardiovascular Genetics|March 12, 2015
Proteomic Analysis of Human Pluripotent Stem Cell-Derived, Fetal, and Adult Ventricular Cardiomyocytes Reveals Pathways Crucial for Cardiac Metabolism and MaturationEllen Poon, Wendy Keung, Yimin Liang, et al.
Cell Reports|December 24, 2016
Integrative Analysis of PRKAG2 Cardiomyopathy iPS and Microtissue Models Identifies AMPK as a Regulator of Metabolism, Survival, and FibrosisJ Travis Hinson, Anant Chopra, Andre Lowe, et al.
Cell Reports|June 15, 2017
Integrative Analysis of PRKAG2 Cardiomyopathy iPS and Microtissue Models Identifies AMPK as a Regulator of Metabolism, Survival, and FibrosisJ Travis Hinson, Anant Chopra, Andre Lowe, et al.
Pageof 3

Showing results (11-20 of 25) with videos related to

Sort By:
Pageof 3
Developmental Cell|January 10, 2018
Force Generation via β-Cardiac Myosin, Titin, and α-Actinin Drives Cardiac Sarcomere Assembly from Cell-Matrix AdhesionsAnant Chopra, Matthew L Kutys, Kehan Zhang, et al.
Nature|May 31, 2023
Transfer learning enables predictions in network biologyChristina V Theodoris, Ling Xiao, Anant Chopra, et al.
Microsystems & Nanoengineering|September 27, 2021
Probing the subcellular nanostructure of engineered human cardiomyocytes in 3D tissueJosh Javor, Jourdan K Ewoldt, Paige E Cloonan, et al.
Circulation Research|August 18, 2021
Filamin C Cardiomyopathy Variants Cause Protein and Lysosome AccumulationRadhika Agarwal, Joao A Paulo, Christopher N Toepfer, et al.
Circulation. Arrhythmia and Electrophysiology|December 16, 2014
Phospholamban as a crucial determinant of the inotropic response of human pluripotent stem cell-derived ventricular cardiomyocytes and engineered 3-dimensional tissue constructsGaopeng Chen, Sen Li, Ioannis Karakikes, et al.
APL Bioengineering|February 16, 2026
Cell shape and maturation impacts α-actinin-2 tension in iPSC-derived cardiomyocytesPalash K Dutta, Joshua M Toth, Subramanian Sundaram, et al.
Circulation Research|February 1, 2019
SarcTrackChristopher N Toepfer, Arun Sharma, Marcelo Cicconet, et al.
Circulation. Cardiovascular Genetics|March 12, 2015
Proteomic Analysis of Human Pluripotent Stem Cell-Derived, Fetal, and Adult Ventricular Cardiomyocytes Reveals Pathways Crucial for Cardiac Metabolism and MaturationEllen Poon, Wendy Keung, Yimin Liang, et al.
Cell Reports|December 24, 2016
Integrative Analysis of PRKAG2 Cardiomyopathy iPS and Microtissue Models Identifies AMPK as a Regulator of Metabolism, Survival, and FibrosisJ Travis Hinson, Anant Chopra, Andre Lowe, et al.
Cell Reports|June 15, 2017
Integrative Analysis of PRKAG2 Cardiomyopathy iPS and Microtissue Models Identifies AMPK as a Regulator of Metabolism, Survival, and FibrosisJ Travis Hinson, Anant Chopra, Andre Lowe, et al.
Pageof 3