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Anastasios V Tzingounis

Showing results (11-20 of 40) with videos related to

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Function (Oxford, England)|May 11, 2023
Differential Control of Small-conductance Calcium-activated Potassium Channel Diffusion by Actin in Different Neuronal SubcompartmentsShiju Gu, Anastasios V Tzingounis, George Lykotrafitis
Frontiers in Cellular Neuroscience|November 1, 2012
The calcium-activated slow AHP: cutting through the Gordian knotRodrigo Andrade, Robert C Foehring, Anastasios V Tzingounis
Journal of Neurophysiology|March 17, 2021
KCNQ3 is the principal target of retigabine in CA1 and subicular excitatory neuronsNissi Varghese, Anna Lauritano, Maurizio Taglialatela, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 11, 2014
Conditional deletions of epilepsy-associated KCNQ2 and KCNQ3 channels from cerebral cortex cause differential effects on neuronal excitabilityHeun Soh, Rima Pant, Joseph J LoTurco, et al.
Biophysical Journal|December 25, 2012
Hippocalcin and KCNQ channels contribute to the kinetics of the slow afterhyperpolarizationKwang S Kim, Masaaki Kobayashi, Ken Takamatsu, et al.
Eneuro|April 17, 2021
Loss of KCNQ2 or KCNQ3 Leads to Multifocal Time-Varying Activity in the Neonatal Forebrain <i>Ex Vivo</i>Bowen Hou, Nissi Varghese, Heun Soh, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 23, 2022
KCNQ2 and KCNQ5 form heteromeric channels independent of KCNQ3Heun Soh, Kristen Springer, Klarita Doci, et al.
Journal of Molecular Biology|April 25, 2016
Tonic PKA Activity Regulates SK Channel Nanoclustering and Somatodendritic DistributionKrithika Abiraman, Megha Sah, Randall S Walikonis, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 20, 2017
Epilepsy-Associated KCNQ2 Channels Regulate Multiple Intrinsic Properties of Layer 2/3 Pyramidal NeuronsZachary Niday, Virginia E Hawkins, Heun Soh, et al.
Elife|November 2, 2018
Deletion of KCNQ2/3 potassium channels from PV+ interneurons leads to homeostatic potentiation of excitatory transmissionHeun Soh, Suhyeorn Park, Kali Ryan, et al.
Pageof 4

Showing results (11-20 of 40) with videos related to

Sort By:
Pageof 4
Function (Oxford, England)|May 11, 2023
Differential Control of Small-conductance Calcium-activated Potassium Channel Diffusion by Actin in Different Neuronal SubcompartmentsShiju Gu, Anastasios V Tzingounis, George Lykotrafitis
Frontiers in Cellular Neuroscience|November 1, 2012
The calcium-activated slow AHP: cutting through the Gordian knotRodrigo Andrade, Robert C Foehring, Anastasios V Tzingounis
Journal of Neurophysiology|March 17, 2021
KCNQ3 is the principal target of retigabine in CA1 and subicular excitatory neuronsNissi Varghese, Anna Lauritano, Maurizio Taglialatela, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 11, 2014
Conditional deletions of epilepsy-associated KCNQ2 and KCNQ3 channels from cerebral cortex cause differential effects on neuronal excitabilityHeun Soh, Rima Pant, Joseph J LoTurco, et al.
Biophysical Journal|December 25, 2012
Hippocalcin and KCNQ channels contribute to the kinetics of the slow afterhyperpolarizationKwang S Kim, Masaaki Kobayashi, Ken Takamatsu, et al.
Eneuro|April 17, 2021
Loss of KCNQ2 or KCNQ3 Leads to Multifocal Time-Varying Activity in the Neonatal Forebrain <i>Ex Vivo</i>Bowen Hou, Nissi Varghese, Heun Soh, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 23, 2022
KCNQ2 and KCNQ5 form heteromeric channels independent of KCNQ3Heun Soh, Kristen Springer, Klarita Doci, et al.
Journal of Molecular Biology|April 25, 2016
Tonic PKA Activity Regulates SK Channel Nanoclustering and Somatodendritic DistributionKrithika Abiraman, Megha Sah, Randall S Walikonis, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 20, 2017
Epilepsy-Associated KCNQ2 Channels Regulate Multiple Intrinsic Properties of Layer 2/3 Pyramidal NeuronsZachary Niday, Virginia E Hawkins, Heun Soh, et al.
Elife|November 2, 2018
Deletion of KCNQ2/3 potassium channels from PV+ interneurons leads to homeostatic potentiation of excitatory transmissionHeun Soh, Suhyeorn Park, Kali Ryan, et al.
Pageof 4