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Iscience|March 15, 2020
Loss of H3K36 Methyltransferase SETD2 Impairs V(D)J Recombination during Lymphoid DevelopmentS Haihua Chu, Jonathan R Chabon, Chloe N Matovina, et al.Nature|February 9, 2012
DNA damage defines sites of recurrent chromosomal translocations in B lymphocytesOfir Hakim, Wolfgang Resch, Arito Yamane, et al.Cell Reports|September 27, 2018
BRCA1 Mutation-Specific Responses to 53BP1 Loss-Induced Homologous Recombination and PARP Inhibitor ResistanceJoseph Nacson, John J Krais, Andrea J Bernhardy, et al.Nature Communications|January 20, 2025
A dual role of Cohesin in DNA DSB repairMichael Fedkenheuer, Yafang Shang, Seolkyoung Jung, et al.Cell|July 1, 2026
Recurrent patterns of TOP1-mediated neuronal genomic damage shared by major neurodegenerative disordersZinan Zhou, Lovelace J Luquette, Guanlan Dong, et al.Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|April 8, 2020
Single-Cell-Derived Primary Rectal Carcinoma Cell Lines Reflect Intratumor Heterogeneity Associated with Treatment ResponseRüdiger Braun, Lena Anthuber, Daniela Hirsch, et al.Biorxiv : the Preprint Server for Biology|June 22, 2026
Mapping the genetic landscape of the DNA damage response with Cas12a-based combinatorial knockout screensSamuel B Hayward, Alina Vaitsiankova, Tomas Lama-Diaz, et al.Biorxiv : the Preprint Server for Biology|July 9, 2024
Neuronal DNA repair reveals strategies to influence CRISPR editing outcomesGokul N Ramadoss, Samali J Namaganda, Jennifer R Hamilton, et al.Elife|June 19, 2024
<i>SOD1</i> is a synthetic-lethal target in <i>PPM1D</i>-mutant leukemia cellsLinda Zhang, Joanne I Hsu, Etienne D Braekeleer, et al.Biorxiv : the Preprint Server for Biology|September 11, 2023
<i>SOD1</i> is a synthetic lethal target in <i>PPM1D</i>-mutant leukemia cellsLinda Zhang, Joanne I Hsu, Etienne D Braekeleer, et al.Pageof 8