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Andreas Friebe

Showing results (51-60 of 75) with videos related to

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Cell Communication and Signaling : CCS|August 13, 2016
Erythrocytes do not activate purified and platelet soluble guanylate cyclases even in conditions favourable for NO synthesisStepan Gambaryan, Hariharan Subramanian, Linda Kehrer, et al.
The Journal of Pharmacology and Experimental Therapeutics|July 10, 2015
Sildenafil Does Not Prevent Heart Hypertrophy and Fibrosis Induced by Cardiomyocyte Angiotensin II Type 1 Receptor SignalingJulia Straubinger, Verena Schöttle, Nadja Bork, et al.
Journal of Theoretical Biology|July 17, 2022
A modular systems biological modelling framework studies cyclic nucleotide signaling in plateletsTim Breitenbach, Nils Englert, Özge Osmanoglu, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 22, 2008
cGMP produced by NO-sensitive guanylyl cyclase essentially contributes to inflammatory and neuropathic pain by using targets different from cGMP-dependent protein kinase IAchim Schmidtko, Wei Gao, Peter König, et al.
Circulation Research|November 19, 2011
Pressure-overload-induced subcellular relocalization/oxidation of soluble guanylyl cyclase in the heart modulates enzyme stimulationEmily J Tsai, Yuchuan Liu, Norimichi Koitabashi, et al.
Nature Communications|November 22, 2018
Publisher Correction: A shear-dependent NO-cGMP-cGKI cascade in platelets acts as an auto-regulatory brake of thrombosisLai Wen, Susanne Feil, Markus Wolters, et al.
Pain|November 14, 2018
Distinct functions of soluble guanylyl cyclase isoforms NO-GC1 and NO-GC2 in inflammatory and neuropathic pain processingJonas Petersen, Evanthia Mergia, Lea Kennel, et al.
Nature Communications|October 18, 2018
A shear-dependent NO-cGMP-cGKI cascade in platelets acts as an auto-regulatory brake of thrombosisLai Wen, Susanne Feil, Markus Wolters, et al.
Hypertension (Dallas, Tex. : 1979)|December 3, 2014
Soluble guanylate cyclase is required for systemic vasodilation but not positive inotropy induced by nitroxyl in the mouseGuangshuo Zhu, Dieter Groneberg, Gautam Sikka, et al.
Nature Communications|May 27, 2015
Stimulation of soluble guanylyl cyclase protects against obesity by recruiting brown adipose tissueLinda S Hoffmann, Jennifer Etzrodt, Lena Willkomm, et al.
Pageof 8

Showing results (51-60 of 75) with videos related to

Sort By:
Pageof 8
Cell Communication and Signaling : CCS|August 13, 2016
Erythrocytes do not activate purified and platelet soluble guanylate cyclases even in conditions favourable for NO synthesisStepan Gambaryan, Hariharan Subramanian, Linda Kehrer, et al.
The Journal of Pharmacology and Experimental Therapeutics|July 10, 2015
Sildenafil Does Not Prevent Heart Hypertrophy and Fibrosis Induced by Cardiomyocyte Angiotensin II Type 1 Receptor SignalingJulia Straubinger, Verena Schöttle, Nadja Bork, et al.
Journal of Theoretical Biology|July 17, 2022
A modular systems biological modelling framework studies cyclic nucleotide signaling in plateletsTim Breitenbach, Nils Englert, Özge Osmanoglu, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 22, 2008
cGMP produced by NO-sensitive guanylyl cyclase essentially contributes to inflammatory and neuropathic pain by using targets different from cGMP-dependent protein kinase IAchim Schmidtko, Wei Gao, Peter König, et al.
Circulation Research|November 19, 2011
Pressure-overload-induced subcellular relocalization/oxidation of soluble guanylyl cyclase in the heart modulates enzyme stimulationEmily J Tsai, Yuchuan Liu, Norimichi Koitabashi, et al.
Nature Communications|November 22, 2018
Publisher Correction: A shear-dependent NO-cGMP-cGKI cascade in platelets acts as an auto-regulatory brake of thrombosisLai Wen, Susanne Feil, Markus Wolters, et al.
Pain|November 14, 2018
Distinct functions of soluble guanylyl cyclase isoforms NO-GC1 and NO-GC2 in inflammatory and neuropathic pain processingJonas Petersen, Evanthia Mergia, Lea Kennel, et al.
Nature Communications|October 18, 2018
A shear-dependent NO-cGMP-cGKI cascade in platelets acts as an auto-regulatory brake of thrombosisLai Wen, Susanne Feil, Markus Wolters, et al.
Hypertension (Dallas, Tex. : 1979)|December 3, 2014
Soluble guanylate cyclase is required for systemic vasodilation but not positive inotropy induced by nitroxyl in the mouseGuangshuo Zhu, Dieter Groneberg, Gautam Sikka, et al.
Nature Communications|May 27, 2015
Stimulation of soluble guanylyl cyclase protects against obesity by recruiting brown adipose tissueLinda S Hoffmann, Jennifer Etzrodt, Lena Willkomm, et al.
Pageof 8