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Experimental & Translational Stroke Medicine
|
December 31, 2013
Ischemia-induced cell depolarization: does the hyperpolarization-activated cation channel HCN2 affect the outcome after stroke in mice?
Petra Ehling, Eva Göb, Stefan Bittner, et al.
The EMBO Journal
|
February 5, 2005
ADAM10 cleavage of N-cadherin and regulation of cell-cell adhesion and beta-catenin nuclear signalling
Karina Reiss, Thorsten Maretzky, Andreas Ludwig, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
August 10, 2007
Dendritic HCN2 channels constrain glutamate-driven excitability in reticular thalamic neurons
Shui-Wang Ying, Fan Jia, Syed Y Abbas, et al.
Circulation Research
|
April 19, 2008
ADAM10 regulates endothelial permeability and T-Cell transmigration by proteolysis of vascular endothelial cadherin
Beate Schulz, Jessica Pruessmeyer, Thorsten Maretzky, et al.
The Journal of Biological Chemistry
|
June 6, 2006
Regulated ADAM10-dependent ectodomain shedding of gamma-protocadherin C3 modulates cell-cell adhesion
Karina Reiss, Thorsten Maretzky, Ingrid G Haas, et al.
Circulation Research
|
February 3, 2007
Importance of CXC chemokine receptor 2 in the homing of human peripheral blood endothelial progenitor cells to sites of arterial injury
Mihail Hristov, Alma Zernecke, Kiril Bidzhekov, et al.
Plos One
|
January 27, 2012
Processing of retinal signals in normal and HCN deficient mice
Luca Della Santina, Ilaria Piano, Lorenzo Cangiano, et al.
Glia
|
May 21, 2009
Involvement of ADAM10 in axonal outgrowth and myelination of the peripheral nerve
Parastoo Jangouk, Thomas Dehmel, Gerd Meyer Zu Hörste, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
December 6, 2003
The hyperpolarization-activated channel HCN4 is required for the generation of pacemaker action potentials in the embryonic heart
Juliane Stieber, Stefan Herrmann, Susanne Feil, et al.
Circulation
|
January 29, 2003
Dominant-negative suppression of HCN channels markedly reduces the native pacemaker current I(f) and undermines spontaneous beating of neonatal cardiomyocytes
Fikret Er, Robert Larbig, Andreas Ludwig, et al.
Page
of 22
Search research articles
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Showing results (71-80 of 212) with videos related to
Sort By:
Page
of 22
Experimental & Translational Stroke Medicine
|
December 31, 2013
Ischemia-induced cell depolarization: does the hyperpolarization-activated cation channel HCN2 affect the outcome after stroke in mice?
Petra Ehling, Eva Göb, Stefan Bittner, et al.
The EMBO Journal
|
February 5, 2005
ADAM10 cleavage of N-cadherin and regulation of cell-cell adhesion and beta-catenin nuclear signalling
Karina Reiss, Thorsten Maretzky, Andreas Ludwig, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
August 10, 2007
Dendritic HCN2 channels constrain glutamate-driven excitability in reticular thalamic neurons
Shui-Wang Ying, Fan Jia, Syed Y Abbas, et al.
Circulation Research
|
April 19, 2008
ADAM10 regulates endothelial permeability and T-Cell transmigration by proteolysis of vascular endothelial cadherin
Beate Schulz, Jessica Pruessmeyer, Thorsten Maretzky, et al.
The Journal of Biological Chemistry
|
June 6, 2006
Regulated ADAM10-dependent ectodomain shedding of gamma-protocadherin C3 modulates cell-cell adhesion
Karina Reiss, Thorsten Maretzky, Ingrid G Haas, et al.
Circulation Research
|
February 3, 2007
Importance of CXC chemokine receptor 2 in the homing of human peripheral blood endothelial progenitor cells to sites of arterial injury
Mihail Hristov, Alma Zernecke, Kiril Bidzhekov, et al.
Plos One
|
January 27, 2012
Processing of retinal signals in normal and HCN deficient mice
Luca Della Santina, Ilaria Piano, Lorenzo Cangiano, et al.
Glia
|
May 21, 2009
Involvement of ADAM10 in axonal outgrowth and myelination of the peripheral nerve
Parastoo Jangouk, Thomas Dehmel, Gerd Meyer Zu Hörste, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
December 6, 2003
The hyperpolarization-activated channel HCN4 is required for the generation of pacemaker action potentials in the embryonic heart
Juliane Stieber, Stefan Herrmann, Susanne Feil, et al.
Circulation
|
January 29, 2003
Dominant-negative suppression of HCN channels markedly reduces the native pacemaker current I(f) and undermines spontaneous beating of neonatal cardiomyocytes
Fikret Er, Robert Larbig, Andreas Ludwig, et al.
Page
of 22