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Brain : a Journal of Neurology|May 29, 2012
C-terminal fragment of N-cadherin accelerates synapse destabilization by amyloid-βAksana Andreyeva, Katja Nieweg, Katharina Horstmann, et al.
International Journal of Molecular Sciences|July 8, 2020
IPSC-Derived Neuronal Cultures Carrying the Alzheimer's Disease Associated TREM2 R47H Variant Enables the Construction of an Aβ-Induced Gene Regulatory NetworkSoraia Martins, Andreas Müller-Schiffmann, Lars Erichsen, et al.
Acta Neuropathologica Communications|August 28, 2016
Revisiting rodent models: Octodon degus as Alzheimer's disease model?Johannes Steffen, Markus Krohn, Kristin Paarmann, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|February 2, 2012
Cellular prion protein participates in amyloid-β transcytosis across the blood-brain barrierThorsten Pflanzner, Benjamin Petsch, Bettina André-Dohmen, et al.
Molecular Immunology|November 1, 2008
Complementarity determining regions of an anti-prion protein scFv fragment orchestrate conformation specificity and antiprion activityAndreas Müller-Schiffmann, Benjamin Petsch, S Rutger Leliveld, et al.
Journal of Virology|February 25, 2011
Biological effects and use of PrPSc- and PrP-specific antibodies generated by immunization with purified full-length native mouse prionsBenjamin Petsch, Andreas Müller-Schiffmann, Anna Lehle, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 11, 2020
Disruption of cellular proteostasis by H1N1 influenza A virus causes α-synuclein aggregationRita Marreiros, Andreas Müller-Schiffmann, Svenja V Trossbach, et al.
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