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EMBO Reports
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April 30, 2025
Transcriptomic changes including p53 dysregulation prime DNMT3A mutant cells for transformation
Erin M Lawrence, Amali Cooray, Andrew J Kueh, et al.
Cell Death & Disease
|
April 26, 2019
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
Erinna F Lee, Tiffany J Harris, Sharon Tran, et al.
Molecular Cell
|
May 22, 2012
The ubiquitin ligase XIAP recruits LUBAC for NOD2 signaling in inflammation and innate immunity
Rune Busk Damgaard, Ueli Nachbur, Monica Yabal, et al.
Nature Medicine
|
June 13, 2018
DNA repair processes are critical mediators of p53-dependent tumor suppression
Ana Janic, Liz J Valente, Matthew J Wakefield, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
July 20, 2011
Fas-mediated neutrophil apoptosis is accelerated by Bid, Bak, and Bax and inhibited by Bcl-2 and Mcl-1
Ben A Croker, Joanne A O'Donnell, Cameron J Nowell, et al.
Cell Death and Differentiation
|
April 1, 2017
Anti-apoptotic proteins BCL-2, MCL-1 and A1 summate collectively to maintain survival of immune cell populations both in vitro and in vivo
Emma M Carrington, Yifan Zhan, Jamie L Brady, et al.
Nature Immunology
|
July 16, 2013
Antiapoptotic Mcl-1 is critical for the survival and niche-filling capacity of Foxp3⁺ regulatory T cells
Wim Pierson, Bénédicte Cauwe, Antonia Policheni, et al.
The Journal of Experimental Medicine
|
January 7, 2017
Cell cycle progression dictates the requirement for BCL2 in natural killer cell survival
Charlotte Viant, Sophie Guia, Robert J Hennessy, et al.
Cell Death and Differentiation
|
October 25, 2019
EBV BCL-2 homologue BHRF1 drives chemoresistance and lymphomagenesis by inhibiting multiple cellular pro-apoptotic proteins
Leah Fitzsimmons, Rachel Cartlidge, Catherine Chang, et al.
Nature Communications
|
March 23, 2025
MCL‑1 safeguards activated hair follicle stem cells to enable adult hair regeneration
Hui San Chin, Jinming Cheng, Shih Han Hsu, et al.
Page
of 32
Search research articles
Search
Showing results (281-290 of 320) with videos related to
Sort By:
Page
of 32
EMBO Reports
|
April 30, 2025
Transcriptomic changes including p53 dysregulation prime DNMT3A mutant cells for transformation
Erin M Lawrence, Amali Cooray, Andrew J Kueh, et al.
Cell Death & Disease
|
April 26, 2019
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
Erinna F Lee, Tiffany J Harris, Sharon Tran, et al.
Molecular Cell
|
May 22, 2012
The ubiquitin ligase XIAP recruits LUBAC for NOD2 signaling in inflammation and innate immunity
Rune Busk Damgaard, Ueli Nachbur, Monica Yabal, et al.
Nature Medicine
|
June 13, 2018
DNA repair processes are critical mediators of p53-dependent tumor suppression
Ana Janic, Liz J Valente, Matthew J Wakefield, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
July 20, 2011
Fas-mediated neutrophil apoptosis is accelerated by Bid, Bak, and Bax and inhibited by Bcl-2 and Mcl-1
Ben A Croker, Joanne A O'Donnell, Cameron J Nowell, et al.
Cell Death and Differentiation
|
April 1, 2017
Anti-apoptotic proteins BCL-2, MCL-1 and A1 summate collectively to maintain survival of immune cell populations both in vitro and in vivo
Emma M Carrington, Yifan Zhan, Jamie L Brady, et al.
Nature Immunology
|
July 16, 2013
Antiapoptotic Mcl-1 is critical for the survival and niche-filling capacity of Foxp3⁺ regulatory T cells
Wim Pierson, Bénédicte Cauwe, Antonia Policheni, et al.
The Journal of Experimental Medicine
|
January 7, 2017
Cell cycle progression dictates the requirement for BCL2 in natural killer cell survival
Charlotte Viant, Sophie Guia, Robert J Hennessy, et al.
Cell Death and Differentiation
|
October 25, 2019
EBV BCL-2 homologue BHRF1 drives chemoresistance and lymphomagenesis by inhibiting multiple cellular pro-apoptotic proteins
Leah Fitzsimmons, Rachel Cartlidge, Catherine Chang, et al.
Nature Communications
|
March 23, 2025
MCL‑1 safeguards activated hair follicle stem cells to enable adult hair regeneration
Hui San Chin, Jinming Cheng, Shih Han Hsu, et al.
Page
of 32