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Molecular Therapy. Nucleic Acids|August 30, 2021
Erratum: LRRK2 Antisense Oligonucleotides Ameliorate α-Synuclein Inclusion Formation in a Parkinson's Disease Mouse ModelHien Tran Zhao, Neena John, Vedad Delic, et al.
Molecular Therapy. Nucleic Acids|June 18, 2021
Erratum: LRRK2 Antisense Oligonucleotides Ameliorate α-Synuclein Inclusion Formation in a Parkinson's Disease Mouse ModelHien Tran Zhao, Neena John, Vedad Delic, et al.
Iscience|January 13, 2022
Discovery of small-molecule positive allosteric modulators of Parkin E3 ligaseEvgeny Shlevkov, Paramasivam Murugan, Dan Montagna, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 30, 2021
Wild-type GBA1 increases the α-synuclein tetramer-monomer ratio, reduces lipid-rich aggregates, and attenuates motor and cognitive deficits in miceKelly E Glajch, Tim E Moors, Yi Chen, et al.
Movement Disorders : Official Journal of the Movement Disorder Society|June 19, 2019
Randomized phase I clinical trial of anti-α-synuclein antibody BIIB054Miroslaw Brys, Laura Fanning, Serena Hung, et al.
Annals of Neurology|September 30, 2020
A Stearoyl-Coenzyme A Desaturase Inhibitor Prevents Multiple Parkinson Disease Phenotypes in α-Synuclein MiceSilke Nuber, Alice Y Nam, Molly M Rajsombath, et al.
Biomedicine & Pharmacotherapy = Biomedecine & Pharmacotherapie|June 24, 2025
Comparative assessment of binding and functional differences of clinical antibodies targeting α-synuclein in cellular models of Parkinson's diseaseYuTing Liu, Isin Dalkilic-Liddle, You Li, et al.
Neurobiology of Disease|November 2, 2018
Development of an aggregate-selective, human-derived α-synuclein antibody BIIB054 that ameliorates disease phenotypes in Parkinson's disease modelsAndreas Weihofen, YuTing Liu, Joseph W Arndt, et al.
Glia|January 29, 2022
Neurodegeneration and neuroinflammation are linked, but independent of alpha-synuclein inclusions, in a seeding/spreading mouse model of Parkinson's diseasePierre Garcia, Wiebke Jürgens-Wemheuer, Oihane Uriarte Huarte, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 14, 2021
The Parkinson's disease-associated gene ITPKB protects against α-synuclein aggregation by regulating ER-to-mitochondria calcium releaseDaniel J Apicco, Evgeny Shlevkov, Catherine L Nezich, et al.
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