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European Journal of Nuclear Medicine and Molecular Imaging
|
December 28, 2020
[<sup>11</sup>C]MODAG-001-towards a PET tracer targeting α-synuclein aggregates
Laura Kuebler, Sabrina Buss, Andrei Leonov, et al.
Nature Communications
|
September 14, 2022
The clinical drug candidate anle138b binds in a cavity of lipidic α-synuclein fibrils
Leif Antonschmidt, Dirk Matthes, Rıza Dervişoğlu, et al.
Ebiomedicine
|
May 2, 2022
Safety, tolerability and pharmacokinetics of the oligomer modulator anle138b with exposure levels sufficient for therapeutic efficacy in a murine Parkinson model: A randomised, double-blind, placebo-controlled phase 1a trial
Johannes Levin, Nand Sing, Sue Melbourne, et al.
Nature Communications
|
October 3, 2025
Anle138b binds predominantly to the central cavity in lipidic Aβ₄₀ fibrils and modulates fibril formation
Mookyoung Han, Benedikt Frieg, Dirk Matthes, et al.
Acta Neuropathologica
|
October 7, 2015
Reducing tau aggregates with anle138b delays disease progression in a mouse model of tauopathies
Jens Wagner, Sybille Krauss, Song Shi, et al.
Alzheimer'S Research & Therapy
|
August 3, 2019
Late-stage Anle138b treatment ameliorates tau pathology and metabolic decline in a mouse model of human Alzheimer's disease tau
Matthias Brendel, Maximilian Deussing, Tanja Blume, et al.
EMBO Molecular Medicine
|
December 7, 2017
The diphenylpyrazole compound anle138b blocks Aβ channels and rescues disease phenotypes in a mouse model for amyloid pathology
Ana Martinez Hernandez, Hendrik Urbanke, Alan L Gillman, et al.
Science Translational Medicine
|
May 27, 2026
The PET tracer [<sup>11</sup>C]MODAG-005 targets alpha-synuclein aggregates in the brain
Ran Sing Saw, Sabrina Haas, Felix Schmidt, et al.
Acta Neuropathologica
|
April 23, 2013
Anle138b: a novel oligomer modulator for disease-modifying therapy of neurodegenerative diseases such as prion and Parkinson's disease
Jens Wagner, Sergey Ryazanov, Andrei Leonov, et al.
Page
of 5
Search research articles
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Showing results (41-50 of 49) with videos related to
Sort By:
Page
of 5
You have reached the last page of results.
This site can display upto 49 results.
European Journal of Nuclear Medicine and Molecular Imaging
|
December 28, 2020
[<sup>11</sup>C]MODAG-001-towards a PET tracer targeting α-synuclein aggregates
Laura Kuebler, Sabrina Buss, Andrei Leonov, et al.
Nature Communications
|
September 14, 2022
The clinical drug candidate anle138b binds in a cavity of lipidic α-synuclein fibrils
Leif Antonschmidt, Dirk Matthes, Rıza Dervişoğlu, et al.
Ebiomedicine
|
May 2, 2022
Safety, tolerability and pharmacokinetics of the oligomer modulator anle138b with exposure levels sufficient for therapeutic efficacy in a murine Parkinson model: A randomised, double-blind, placebo-controlled phase 1a trial
Johannes Levin, Nand Sing, Sue Melbourne, et al.
Nature Communications
|
October 3, 2025
Anle138b binds predominantly to the central cavity in lipidic Aβ₄₀ fibrils and modulates fibril formation
Mookyoung Han, Benedikt Frieg, Dirk Matthes, et al.
Acta Neuropathologica
|
October 7, 2015
Reducing tau aggregates with anle138b delays disease progression in a mouse model of tauopathies
Jens Wagner, Sybille Krauss, Song Shi, et al.
Alzheimer'S Research & Therapy
|
August 3, 2019
Late-stage Anle138b treatment ameliorates tau pathology and metabolic decline in a mouse model of human Alzheimer's disease tau
Matthias Brendel, Maximilian Deussing, Tanja Blume, et al.
EMBO Molecular Medicine
|
December 7, 2017
The diphenylpyrazole compound anle138b blocks Aβ channels and rescues disease phenotypes in a mouse model for amyloid pathology
Ana Martinez Hernandez, Hendrik Urbanke, Alan L Gillman, et al.
Science Translational Medicine
|
May 27, 2026
The PET tracer [<sup>11</sup>C]MODAG-005 targets alpha-synuclein aggregates in the brain
Ran Sing Saw, Sabrina Haas, Felix Schmidt, et al.
Acta Neuropathologica
|
April 23, 2013
Anle138b: a novel oligomer modulator for disease-modifying therapy of neurodegenerative diseases such as prion and Parkinson's disease
Jens Wagner, Sergey Ryazanov, Andrei Leonov, et al.
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of 5