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Visual Neuroscience|April 17, 2010
V1 orientation plasticity is explained by broadly tuned feedforward inputs and intracortical sharpeningAndrew F Teich, Ning QianJournal of Neurophysiology|March 4, 2003
Learning and adaptation in a recurrent model of V1 orientation selectivityAndrew F Teich, Ning QianJournal of Neurophysiology|April 21, 2006
Comparison among some models of orientation selectivityAndrew F Teich, Ning QianThe Biochemical Journal|August 16, 2012
Is the amyloid hypothesis of Alzheimer's disease therapeutically relevant?Andrew F Teich, Ottavio ArancioPlos One|February 6, 2013
A reliable way to detect endogenous murine β-amyloidAndrew F Teich, Mitesh Patel, Ottavio ArancioPlos One|May 11, 2026
Comparative genomics of human brain and immune gene preservation across speciesXiao Liang, Andrew F Teich, Lenwood S HeathFrontiers in Aging Neuroscience|February 26, 2026
ZCCHC17: a target for synaptic dysfunction and neuronal excitability in Alzheimer's diseaseBrittany A Klub, Andrew F Teich, Giuseppe P CorteseBioinformatics (Oxford, England)|October 14, 2017
ZCCHC17 is a master regulator of synaptic gene expression in Alzheimer's diseaseZeljko Tomljanovic, Mitesh Patel, William Shin, et al.Free Neuropathology|June 22, 2023
Survey of Neuroanatomic Sampling and Staining Procedures in Alzheimer Disease Research Center Brain BanksJuan C Vizcarra, Andrew F Teich, Brittany N Dugger, et al.Journal of Alzheimer'S Disease : JAD|July 20, 2012
5-HT₄ receptor stimulation leads to soluble AβPPα production through MMP-9 upregulationGakuji Hashimoto, Mikako Sakurai, Andrew F Teich, et al.Pageof 20