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Open Biology|November 17, 2017
Soluble Aβ aggregates can inhibit prion propagationClaire J Sarell, Emma Quarterman, Daniel C-M Yip, et al.
Plos One|November 17, 2023
Two mouse models of Alzheimer's disease accumulate amyloid at different rates and have distinct Aβ oligomer profiles unaltered by ablation of cellular prion proteinSilvia A Purro, Michael Farmer, Elizabeth Noble, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 3, 2014
Peripheral administration of a humanized anti-PrP antibody blocks Alzheimer's disease Aβ synaptotoxicityIgor Klyubin, Andrew J Nicoll, Azadeh Khalili-Shirazi, et al.
Nature|September 11, 2015
Evidence for human transmission of amyloid-β pathology and cerebral amyloid angiopathyZane Jaunmuktane, Simon Mead, Matthew Ellis, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 30, 2010
Pharmacological chaperone for the structured domain of human prion proteinAndrew J Nicoll, Clare R Trevitt, M Howard Tattum, et al.
Nature Communications|September 12, 2013
Amyloid-β nanotubes are associated with prion protein-dependent synaptotoxicityAndrew J Nicoll, Silvia Panico, Darragh B Freir, et al.
Nature Communications|June 10, 2011
Interaction between prion protein and toxic amyloid β assemblies can be therapeutically targeted at multiple sitesDarragh B Freir, Andrew J Nicoll, Igor Klyubin, et al.
The Journal of Biological Chemistry|July 31, 2014
N-terminal domain of prion protein directs its oligomeric associationClare R Trevitt, Laszlo L P Hosszu, Mark Batchelor, et al.
Science (New York, N.Y.)|August 9, 2014
C9orf72 repeat expansions cause neurodegeneration in Drosophila through arginine-rich proteinsSarah Mizielinska, Sebastian Grönke, Teresa Niccoli, et al.
EMBO Molecular Medicine|November 9, 2017
G-quadruplex-binding small molecules ameliorate <i>C9orf72</i> FTD/ALS pathology <i>in vitro</i> and <i>in vivo</i>Roberto Simone, Rubika Balendra, Thomas G Moens, et al.
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