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Andrew Leask

Showing results (171-180 of 221) with videos related to

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Rheumatology (Oxford, England)|September 15, 2012
Focal adhesion kinase and reactive oxygen species contribute to the persistent fibrotic phenotype of lesional scleroderma fibroblastsXu Shi-wen, Katherine Thompson, Korsa Khan, et al.
The Journal of Clinical Investigation|September 20, 2008
GSK-3beta in mouse fibroblasts controls wound healing and fibrosis through an endothelin-1-dependent mechanismMohit Kapoor, Shangxi Liu, Xu Shi-wen, et al.
Journal of Cell Science|January 24, 2012
Periostin modulates myofibroblast differentiation during full-thickness cutaneous wound repairChristopher G Elliott, Jian Wang, Xiaolei Guo, et al.
Annals of the Rheumatic Diseases|June 22, 2018
Connective tissue growth factor contributes to joint homeostasis and osteoarthritis severity by controlling the matrix sequestration and activation of latent TGFβXiaodi Tang, Hayat Muhammad, Celia McLean, et al.
The Journal of Investigative Dermatology|June 10, 2011
Loss of PTEN expression by dermal fibroblasts causes skin fibrosisSunil K Parapuram, Xu Shi-wen, Christopher Elliott, et al.
Arthritis and Rheumatism|January 1, 2009
Requirement of transforming growth factor beta-activated kinase 1 for transforming growth factor beta-induced alpha-smooth muscle actin expression and extracellular matrix contraction in fibroblastsXu Shi-wen, Sunil K Parapuram, Daphne Pala, et al.
Fibrogenesis & Tissue Repair|April 2, 2011
Thrombospondin 1 is a key mediator of transforming growth factor β-mediated cell contractility in systemic sclerosis via a mitogen-activated protein kinase kinase (MEK)/extracellular signal-regulated kinase (ERK)-dependent mechanismYunliang Chen, Andrew Leask, David J Abraham, et al.
Fibrogenesis & Tissue Repair|March 17, 2015
Erratum to: Thrombospondin 1 is a key mediator of transforming growth factor b-mediated cell contractility in systemic sclerosis via a mitogen-activated protein kinase kinase (MEK)/extracellular signal-regulated kinase (ERK)-dependent mechanismYunliang Chen, Andrew Leask, David J Abraham, et al.
Journal of the American Society of Nephrology : JASN|June 20, 2020
Epithelial Vasopressin Type-2 Receptors Regulate Myofibroblasts by a YAP-CCN2-Dependent Mechanism in Polycystic Kidney DiseaseNidhi Dwivedi, Shixin Tao, Abeda Jamadar, et al.
Cancer Research Communications|February 16, 2024
Cancer-associated Fibroblast-specific Expression of the Matricellular Protein CCN1 Coordinates Neovascularization and Stroma Deposition in Melanoma MetastasisJames Hutchenreuther, John Nguyen, Katherine Quesnel, et al.
Pageof 23

Showing results (171-180 of 221) with videos related to

Sort By:
Pageof 23
Rheumatology (Oxford, England)|September 15, 2012
Focal adhesion kinase and reactive oxygen species contribute to the persistent fibrotic phenotype of lesional scleroderma fibroblastsXu Shi-wen, Katherine Thompson, Korsa Khan, et al.
The Journal of Clinical Investigation|September 20, 2008
GSK-3beta in mouse fibroblasts controls wound healing and fibrosis through an endothelin-1-dependent mechanismMohit Kapoor, Shangxi Liu, Xu Shi-wen, et al.
Journal of Cell Science|January 24, 2012
Periostin modulates myofibroblast differentiation during full-thickness cutaneous wound repairChristopher G Elliott, Jian Wang, Xiaolei Guo, et al.
Annals of the Rheumatic Diseases|June 22, 2018
Connective tissue growth factor contributes to joint homeostasis and osteoarthritis severity by controlling the matrix sequestration and activation of latent TGFβXiaodi Tang, Hayat Muhammad, Celia McLean, et al.
The Journal of Investigative Dermatology|June 10, 2011
Loss of PTEN expression by dermal fibroblasts causes skin fibrosisSunil K Parapuram, Xu Shi-wen, Christopher Elliott, et al.
Arthritis and Rheumatism|January 1, 2009
Requirement of transforming growth factor beta-activated kinase 1 for transforming growth factor beta-induced alpha-smooth muscle actin expression and extracellular matrix contraction in fibroblastsXu Shi-wen, Sunil K Parapuram, Daphne Pala, et al.
Fibrogenesis & Tissue Repair|April 2, 2011
Thrombospondin 1 is a key mediator of transforming growth factor β-mediated cell contractility in systemic sclerosis via a mitogen-activated protein kinase kinase (MEK)/extracellular signal-regulated kinase (ERK)-dependent mechanismYunliang Chen, Andrew Leask, David J Abraham, et al.
Fibrogenesis & Tissue Repair|March 17, 2015
Erratum to: Thrombospondin 1 is a key mediator of transforming growth factor b-mediated cell contractility in systemic sclerosis via a mitogen-activated protein kinase kinase (MEK)/extracellular signal-regulated kinase (ERK)-dependent mechanismYunliang Chen, Andrew Leask, David J Abraham, et al.
Journal of the American Society of Nephrology : JASN|June 20, 2020
Epithelial Vasopressin Type-2 Receptors Regulate Myofibroblasts by a YAP-CCN2-Dependent Mechanism in Polycystic Kidney DiseaseNidhi Dwivedi, Shixin Tao, Abeda Jamadar, et al.
Cancer Research Communications|February 16, 2024
Cancer-associated Fibroblast-specific Expression of the Matricellular Protein CCN1 Coordinates Neovascularization and Stroma Deposition in Melanoma MetastasisJames Hutchenreuther, John Nguyen, Katherine Quesnel, et al.
Pageof 23