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American Journal of Physiology. Heart and Circulatory Physiology|September 28, 2014
An acute rise in intraluminal pressure shifts the mediator of flow-mediated dilation from nitric oxide to hydrogen peroxide in human arteriolesAndreas M Beyer, Matthew J Durand, Joseph Hockenberry, et al.
American Journal of Hypertension|February 17, 2012
Nitric oxide synthase-dependent vasodilation of human subcutaneous arterioles correlates with noninvasive measurements of endothelial functionKodlipet Dharmashankar, Aimee Welsh, Jingli Wang, et al.
Microcirculation (New York, N.Y. : 1994)|November 22, 2017
Shaker-related voltage-gated K+ channel expression and vasomotor function in human coronary resistance arteriesYoshinori Nishijima, Ankush Korishettar, Dawid S Chabowski, et al.
International Journal of Cardiology|May 19, 2009
Systemic and microvascular oxidative stress induced by light chain amyloidosisRaymond Q Migrino, Parameswaran Hari, David D Gutterman, et al.
American Journal of Physiology. Heart and Circulatory Physiology|April 18, 2006
Protective effects of epoxyeicosatrienoic acids on human endothelial cells from the pulmonary and coronary vasculatureAnuradha Dhanasekaran, Rula Al-Saghir, Bernardo Lopez, et al.
Anesthesiology|March 26, 2008
Age-related attenuation of isoflurane preconditioning in human atrial cardiomyocytes: roles for mitochondrial respiration and sarcolemmal adenosine triphosphate-sensitive potassium channel activityYasushi Mio, Martin W Bienengraeber, Jasna Marinovic, et al.
Chest|February 9, 2012
Introduction to the ninth edition: Antithrombotic Therapy and Prevention of Thrombosis, 9th ed: American College of Chest Physicians Evidence-Based Clinical Practice GuidelinesGordon H Guyatt, Elie A Akl, Mark Crowther, et al.
Circulation Research|December 14, 2011
H2O2-induced dilation in human coronary arterioles: role of protein kinase G dimerization and large-conductance Ca2+-activated K+ channel activationDavid X Zhang, Lena Borbouse, Debebe Gebremedhin, et al.
American Journal of Physiology. Heart and Circulatory Physiology|February 19, 2017
Mitochondria-regulated formation of endothelium-derived extracellular vesicles shifts the mediator of flow-induced vasodilationJulie K Freed, Matthew J Durand, Brian R Hoffmann, et al.
Basic Research in Cardiology|November 2, 2025
The role of NO, H2O2, and non-NO/H2O2 mechanisms in acetylcholine (ACh)-induced dilation of human arterioles in the absence and presence of coronary artery diseaseKostiantyn Drachuk, Yoshinori Nishijima, Aravind Parthasarathy, et al.
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