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Angela Gomez-Arboledas

Showing results (1-10 of 31) with videos related to

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Frontiers in Aging Neuroscience|September 29, 2023
Editorial: Expert opinions on the relationship between microglia, glia, and Alzheimer's disease: 2022Angela Gomez-Arboledas, Helene Eliane Hirbec
Advances in Immunology|November 30, 2021
Complement as a powerful "influencer" in the brain during development, adulthood and neurological disordersTiffany J Petrisko, Angela Gomez-Arboledas, Andrea J Tenner
Immunotargets and Therapy|October 1, 2021
The Role of Complement in Synaptic Pruning and NeurodegenerationAngela Gomez-Arboledas, Munjal M Acharya, Andrea J Tenner
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 24, 2025
Basic Science and PathogenesisTiffany J Petrisko, Angela Gomez-Arboledas, Shu-Hui Chu, et al.
Biorxiv : the Preprint Server for Biology|September 26, 2025
Young adult microglial deletion of C1q reduces engulfment of synapses and prevents cognitive impairment in an aggressive Alzheimer's disease mouse modelTiffany J Petrisko, Shu-Hui Chu, Angela Gomez-Arboledas, et al.
Current Alzheimer Research|September 1, 2019
Astrocytes: From the Physiology to the DiseaseLaura Trujillo-Estrada, Angela Gomez-Arboledas, Stefânia Forner, et al.
Biorxiv : the Preprint Server for Biology|October 24, 2023
C5aR1 signaling promotes region and age dependent synaptic pruning in models of Alzheimer's DiseaseAngela Gomez-Arboledas, Maria I Fonseca, Enikö Kramar, et al.
Journal of Alzheimer'S Disease : JAD|June 15, 2014
Early neuronal loss and axonal/presynaptic damage is associated with accelerated amyloid-β accumulation in AβPP/PS1 Alzheimer's disease mice subiculumLaura Trujillo-Estrada, José Carlos Dávila, Elisabeth Sánchez-Mejias, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|January 26, 2024
C5aR1 signaling promotes region- and age-dependent synaptic pruning in models of Alzheimer's diseaseAngela Gomez-Arboledas, Maria I Fonseca, Enikö Kramar, et al.
Nature Communications|August 15, 2024
C5aR1 antagonism suppresses inflammatory glial responses and alters cellular signaling in an Alzheimer's disease mouse modelNicole D Schartz, Heidi Y Liang, Klebea Carvalho, et al.
Pageof 4

Showing results (1-10 of 31) with videos related to

Sort By:
Pageof 4
Frontiers in Aging Neuroscience|September 29, 2023
Editorial: Expert opinions on the relationship between microglia, glia, and Alzheimer's disease: 2022Angela Gomez-Arboledas, Helene Eliane Hirbec
Advances in Immunology|November 30, 2021
Complement as a powerful "influencer" in the brain during development, adulthood and neurological disordersTiffany J Petrisko, Angela Gomez-Arboledas, Andrea J Tenner
Immunotargets and Therapy|October 1, 2021
The Role of Complement in Synaptic Pruning and NeurodegenerationAngela Gomez-Arboledas, Munjal M Acharya, Andrea J Tenner
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 24, 2025
Basic Science and PathogenesisTiffany J Petrisko, Angela Gomez-Arboledas, Shu-Hui Chu, et al.
Biorxiv : the Preprint Server for Biology|September 26, 2025
Young adult microglial deletion of C1q reduces engulfment of synapses and prevents cognitive impairment in an aggressive Alzheimer's disease mouse modelTiffany J Petrisko, Shu-Hui Chu, Angela Gomez-Arboledas, et al.
Current Alzheimer Research|September 1, 2019
Astrocytes: From the Physiology to the DiseaseLaura Trujillo-Estrada, Angela Gomez-Arboledas, Stefânia Forner, et al.
Biorxiv : the Preprint Server for Biology|October 24, 2023
C5aR1 signaling promotes region and age dependent synaptic pruning in models of Alzheimer's DiseaseAngela Gomez-Arboledas, Maria I Fonseca, Enikö Kramar, et al.
Journal of Alzheimer'S Disease : JAD|June 15, 2014
Early neuronal loss and axonal/presynaptic damage is associated with accelerated amyloid-β accumulation in AβPP/PS1 Alzheimer's disease mice subiculumLaura Trujillo-Estrada, José Carlos Dávila, Elisabeth Sánchez-Mejias, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|January 26, 2024
C5aR1 signaling promotes region- and age-dependent synaptic pruning in models of Alzheimer's diseaseAngela Gomez-Arboledas, Maria I Fonseca, Enikö Kramar, et al.
Nature Communications|August 15, 2024
C5aR1 antagonism suppresses inflammatory glial responses and alters cellular signaling in an Alzheimer's disease mouse modelNicole D Schartz, Heidi Y Liang, Klebea Carvalho, et al.
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