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Oxidative Medicine and Cellular Longevity|July 29, 2017
Chk1 Promotes DNA Damage Response Bypass following Oxidative Stress in a Model of Hydrogen Peroxide-Associated Ulcerative Colitis through JNK Inactivation and Chromatin BindingKathrin Reissig, Andrew Silver, Roland Hartig, et al.The American Journal of Pathology|July 25, 2009
RETRACTED: Identification of phosphorylated p38 as a novel DAPK-interacting partner during TNFalpha-induced apoptosis in colorectal tumor cellsKhuloud Bajbouj, Angela Poehlmann, Doerthe Kuester, et al.Inflammatory Bowel Diseases|February 5, 2014
DUOX2 and DUOXA2 form the predominant enzyme system capable of producing the reactive oxygen species H2O2 in active ulcerative colitis and are modulated by 5-aminosalicylic acidTammie S MacFie, Richard Poulsom, Alexandra Parker, et al.Pageof 2