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Angelita Tottene

Showing results (1-10 of 15) with videos related to

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Channels (Austin, Tex.)|December 15, 2010
Role of different voltage-gated Ca2+ channels in cortical spreading depression: specific requirement of P/Q-type Ca2+ channelsAngelita Tottene, Andrea Urbani, Daniela Pietrobon
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 25, 2019
Enhanced Thalamocortical Synaptic Transmission and Dysregulation of the Excitatory-Inhibitory Balance at the Thalamocortical Feedforward Inhibitory Microcircuit in a Genetic Mouse Model of MigraineAngelita Tottene, Morgana Favero, Daniela Pietrobon
The Journal of Headache and Pain|November 13, 2025
Mechanisms underlying the increased susceptibility to initiation of cortical spreading depression in a genetic mouse model of migraineMarina Vitale, Angelita Tottene, Maral Zarin Zadeh, et al.
The Journal of Headache and Pain|August 8, 2023
Mechanisms of initiation of cortical spreading depressionMarina Vitale, Angelita Tottene, Maral Zarin Zadeh, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 21, 2022
Enhanced Feedback Inhibition Due to Increased Recruitment of Somatostatin-Expressing Interneurons and Enhanced Cortical Recurrent Excitation in a Genetic Mouse Model of MigraineIvan Marchionni, Nadia Pilati, Angelo Forli, et al.
Frontiers in Cellular Neuroscience|March 6, 2015
Abnormal cortical synaptic transmission in CaV2.1 knockin mice with the S218L missense mutation which causes a severe familial hemiplegic migraine syndrome in humansDania Vecchia, Angelita Tottene, Arn M J M van den Maagdenberg, et al.
Neurobiology of Disease|June 8, 2014
Mechanism underlying unaltered cortical inhibitory synaptic transmission in contrast with enhanced excitatory transmission in CaV2.1 knockin migraine miceDania Vecchia, Angelita Tottene, Arn M J M van den Maagdenberg, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 18, 2002
Familial hemiplegic migraine mutations increase Ca(2+) influx through single human CaV2.1 channels and decrease maximal CaV2.1 current density in neuronsAngelita Tottene, Tommaso Fellin, Stefano Pagnutti, et al.
Neurobiology of Disease|June 10, 2021
Specific activation of GluN1-N2B NMDA receptors underlies facilitation of cortical spreading depression in a genetic mouse model of migraine with reduced astrocytic glutamate clearanceGiovanna Crivellaro, Angelita Tottene, Marina Vitale, et al.
The Journal of Headache and Pain|September 29, 2022
Synaptic alterations in visual cortex reshape contrast-dependent gamma oscillations and inhibition-excitation ratio in a genetic mouse model of migraineNicolò Meneghetti, Chiara Cerri, Eleonora Vannini, et al.
Pageof 2

Showing results (1-10 of 15) with videos related to

Sort By:
Pageof 2
Channels (Austin, Tex.)|December 15, 2010
Role of different voltage-gated Ca2+ channels in cortical spreading depression: specific requirement of P/Q-type Ca2+ channelsAngelita Tottene, Andrea Urbani, Daniela Pietrobon
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 25, 2019
Enhanced Thalamocortical Synaptic Transmission and Dysregulation of the Excitatory-Inhibitory Balance at the Thalamocortical Feedforward Inhibitory Microcircuit in a Genetic Mouse Model of MigraineAngelita Tottene, Morgana Favero, Daniela Pietrobon
The Journal of Headache and Pain|November 13, 2025
Mechanisms underlying the increased susceptibility to initiation of cortical spreading depression in a genetic mouse model of migraineMarina Vitale, Angelita Tottene, Maral Zarin Zadeh, et al.
The Journal of Headache and Pain|August 8, 2023
Mechanisms of initiation of cortical spreading depressionMarina Vitale, Angelita Tottene, Maral Zarin Zadeh, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 21, 2022
Enhanced Feedback Inhibition Due to Increased Recruitment of Somatostatin-Expressing Interneurons and Enhanced Cortical Recurrent Excitation in a Genetic Mouse Model of MigraineIvan Marchionni, Nadia Pilati, Angelo Forli, et al.
Frontiers in Cellular Neuroscience|March 6, 2015
Abnormal cortical synaptic transmission in CaV2.1 knockin mice with the S218L missense mutation which causes a severe familial hemiplegic migraine syndrome in humansDania Vecchia, Angelita Tottene, Arn M J M van den Maagdenberg, et al.
Neurobiology of Disease|June 8, 2014
Mechanism underlying unaltered cortical inhibitory synaptic transmission in contrast with enhanced excitatory transmission in CaV2.1 knockin migraine miceDania Vecchia, Angelita Tottene, Arn M J M van den Maagdenberg, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 18, 2002
Familial hemiplegic migraine mutations increase Ca(2+) influx through single human CaV2.1 channels and decrease maximal CaV2.1 current density in neuronsAngelita Tottene, Tommaso Fellin, Stefano Pagnutti, et al.
Neurobiology of Disease|June 10, 2021
Specific activation of GluN1-N2B NMDA receptors underlies facilitation of cortical spreading depression in a genetic mouse model of migraine with reduced astrocytic glutamate clearanceGiovanna Crivellaro, Angelita Tottene, Marina Vitale, et al.
The Journal of Headache and Pain|September 29, 2022
Synaptic alterations in visual cortex reshape contrast-dependent gamma oscillations and inhibition-excitation ratio in a genetic mouse model of migraineNicolò Meneghetti, Chiara Cerri, Eleonora Vannini, et al.
Pageof 2