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Cardiovascular Research|November 15, 2017
Local production of tenascin-C acts as a trigger for monocyte/macrophage recruitment that provokes cardiac dysfunctionDounia Abbadi, Fanny Laroumanie, Mathilde Bizou, et al.Elife|December 10, 2019
Vasohibin1, a new mouse cardiomyocyte IRES trans-acting factor that regulates translation in early hypoxiaFransky Hantelys, Anne-Claire Godet, Florian David, et al.Cell Death and Differentiation|December 11, 2019
Mitochondrial 4-HNE derived from MAO-A promotes mitoCa<sup>2+</sup> overload in chronic postischemic cardiac remodelingYohan Santin, Loubina Fazal, Yannis Sainte-Marie, et al.Theranostics|November 1, 2023
Inhalation of acidic nanoparticles prevents doxorubicin cardiotoxicity through improvement of lysosomal functionYohan Santin, Karina Formoso, Fraha Haidar, et al.Antioxidants & Redox Signaling|June 29, 2012
p53-PGC-1α pathway mediates oxidative mitochondrial damage and cardiomyocyte necrosis induced by monoamine oxidase-A upregulation: role in chronic left ventricular dysfunction in miceChristelle Villeneuve, Céline Guilbeau-Frugier, Pierre Sicard, et al.Science Advances|December 13, 2024
Computational and digital analyses in the INSPIRE mouse cohort to define sex-specific functional determinants of biological agingYohan Santin, Mattia Chiesa, Amélie Alfonso, et al.Journal of Cachexia, Sarcopenia and Muscle|January 14, 2024
Ryanodine receptor dysfunction causes senescence and fibrosis in Duchenne dilated cardiomyopathyMonia Souidi, Jessica Resta, Haikel Dridi, et al.European Heart Journal|December 4, 2010
Intracoronary autologous mononucleated bone marrow cell infusion for acute myocardial infarction: results of the randomized multicenter BONAMI trialJérôme Roncalli, Frédéric Mouquet, Christophe Piot, et al.The EMBO Journal|February 10, 2019
Length-independent telomere damage drives post-mitotic cardiomyocyte senescenceRhys Anderson, Anthony Lagnado, Damien Maggiorani, et al.Pageof 11