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Scientific Reports|February 16, 2019
Three distinct regions of cRaf kinase domain interact with membranePriyanka Prakash, John F Hancock, Alemayehu A GorfeBiophysical Journal|August 19, 2009
On the use of Ripley's K-function and its derivatives to analyze domain sizeMaria A Kiskowski, John F Hancock, Anne K KenworthyAnti-Cancer Drugs|September 13, 2023
Cellular responses after (neratinib plus pemetrexed) exposure in NSCLC cellsLaurence Booth, Andrew Poklepovic, John F Hancock, et al.Journal of Cell Science|February 24, 2006
Biogenesis of caveolae: a structural model for caveolin-induced domain formationRobert G Parton, Michael Hanzal-Bayer, John F HancockMolecular Membrane Biology|December 31, 2008
Ras acylation, compartmentalization and signaling nanoclusters (Review)Yoav I Henis, John F Hancock, Ian A PriorCommunicative & Integrative Biology|August 30, 2013
Staurosporine: A new tool for studying phosphatidylserine traffickingKwang-Jin Cho, Jin-Hee Park, John F HancockMolecular Cell|March 29, 2023
Consensus on the RAS dimerization hypothesis: Strong evidence for lipid-mediated clustering but not for G-domain-mediated interactionsDhirendra K Simanshu, Mark R Philips, John F HancockCommunicative & Integrative Biology|June 30, 2010
Nucleophosmin and nucleolin regulate K-Ras signalingKerry L Inder, Michelle M Hill, John F HancockCancer Drug Resistance (Alhambra, Calif.)|May 18, 2022
Kinase inhibitors: look beyond the label on the bottlePaul Dent, Andrew Poklepovic, Laurence Booth, et al.Cellular Signalling|September 15, 2019
Signaling alterations caused by drugs and autophagyPaul Dent, Laurence Booth, Andrew Poklepovic, et al.Pageof 17