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Journal of Cellular Physiology|January 9, 2020
Neratinib degrades MST4 via autophagy that reduces membrane stiffness and is essential for the inactivation of PI3K, ERK1/2, and YAP/TAZ signalingPaul Dent, Laurence Booth, Andrew Poklepovic, et al.Cell|January 3, 2017
Lipid-Sorting Specificity Encoded in K-Ras Membrane Anchor Regulates Signal OutputYong Zhou, Priyanka Prakash, Hong Liang, et al.Nature Communications|September 10, 2021
Oncogenic KRAS is dependent upon an EFR3A-PI4KA signaling axis for potent tumorigenic activityHema Adhikari, Walaa E Kattan, Shivesh Kumar, et al.The Journal of Physical Chemistry. B|April 30, 2021
Monoubiquitination of KRAS at Lysine104 and Lysine147 Modulates Its Dynamics and Interaction with Partner ProteinsVinay V Nair, Guowei Yin, Jerry Zhang, et al.Molecular Biology of the Cell|September 12, 2008
Activation of the MAPK module from different spatial locations generates distinct system outputsKerry Inder, Angus Harding, Sarah J Plowman, et al.Scientific Reports|November 4, 2016
Epac1 interacts with importin β1 and controls neurite outgrowth independently of cAMP and Rap1Faiza Baameur, Pooja Singhmar, Yong Zhou, et al.Molecular and Cellular Biology|January 20, 2011
Therapeutic levels of the hydroxmethylglutaryl-coenzyme A reductase inhibitor lovastatin activate ras signaling via phospholipase D2Kwang-Jin Cho, Michelle M Hill, Sravanthi Chigurupati, et al.Traffic (Copenhagen, Denmark)|April 28, 2007
Cholesterol-sensitive Cdc42 activation regulates actin polymerization for endocytosis via the GEEC pathwayRahul Chadda, Mark T Howes, Sarah J Plowman, et al.Iscience|July 5, 2021
p53 mitigates the effects of oncogenic HRAS in urothelial cells via the repression of MCOLN1Jewon Jung, Han Liao, Shannon A Coker, et al.Biochemical Pharmacology|June 2, 2021
Osimertinib-resistant NSCLC cells activate ERBB2 and YAP/TAZ and are killed by neratinibPaul Dent, Laurence Booth, Andrew Poklepovic, et al.Pageof 16