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Molecular and Cellular Biology|April 3, 2004
How many mutant p53 molecules are needed to inactivate a tetramer?Wan Mui Chan, Wai Yi Siu, Anita Lau, et al.The Journal of Biological Chemistry|July 18, 2002
Cyclin F is degraded during G2-M by mechanisms fundamentally different from other cyclinsTsz Kan Fung, Wai Yi Siu, Cain H Yam, et al.Cancer Research|February 21, 2006
Stalled replication induces p53 accumulation through distinct mechanisms from DNA damage checkpoint pathwaysChui Chui Ho, Wai Yi Siu, Anita Lau, et al.FEBS Letters|October 24, 2003
ING1b decreases cell proliferation through p53-dependent and -independent mechanismsFan Cheung Tsang, Lai See Po, Ka Man Leung, et al.Molecular Cancer Research : MCR|February 1, 2006
Ubiquitination of p53 at multiple sites in the DNA-binding domainWan Mui Chan, Man Chi Mak, Tsz Kan Fung, et al.Molecular Cancer Therapeutics|May 14, 2004
Topoisomerase poisons differentially activate DNA damage checkpoints through ataxia-telangiectasia mutated-dependent and -independent mechanismsWai Yi Siu, Anita Lau, Talha Arooz, et al.The Journal of Biological Chemistry|December 19, 2003
Differential mode of regulation of the checkpoint kinases CHK1 and CHK2 by their regulatory domainsChuen-Pei Ng, Hung Chiu Lee, Chung Wai Ho, et al.Cancer Research|September 5, 2002
The candidate tumor suppressor ING1b can stabilize p53 by disrupting the regulation of p53 by MDM2Ka Man Leung, Lai See Po, Fan Cheung Tsang, et al.Experimental Cell Research|February 15, 2005
The relative contribution of CHK1 and CHK2 to Adriamycin-induced checkpointChui Chui Ho, Wai Yi Siu, Jeremy P H Chow, et al.Molecular Biology of the Cell|October 1, 2003
DNA damage during the spindle-assembly checkpoint degrades CDC25A, inhibits cyclin-CDC2 complexes, and reverses cells to interphaseJeremy P H Chow, Wai Yi Siu, Tsz Kan Fung, et al.Pageof 1