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Hematology/Oncology Clinics of North America|September 27, 2012
Myeloproliferative neoplasm animal modelsAnn Mullally, Steven W Lane, Kristina Brumme, et al.Cell Stem Cell|September 10, 2013
Sinister symbiosis: pathological hematopoietic-stromal interactions in CMLAnn Mullally, Benjamin L EbertClinical Cancer Research : an Official Journal of the American Association for Cancer Research|July 1, 2010
NF1 inactivation revs up Ras in adult acute myelogenous leukemiaAnn Mullally, Benjamin L EbertBlood|May 26, 2012
Distinct roles for long-term hematopoietic stem cells and erythroid precursor cells in a murine model of Jak2V617F-mediated polycythemia veraAnn Mullally, Luke Poveromo, Rebekka K Schneider, et al.Cancers|August 27, 2020
Murine Models of MyelofibrosisSebastien Jacquelin, Frederike Kramer, Ann Mullally, et al.Blood|October 5, 2014
Distinct effects of concomitant Jak2V617F expression and Tet2 loss in mice promote disease progression in myeloproliferative neoplasmsEdwin Chen, Rebekka K Schneider, Lawrence J Breyfogle, et al.Cancer Cell|June 15, 2010
Physiological Jak2V617F expression causes a lethal myeloproliferative neoplasm with differential effects on hematopoietic stem and progenitor cellsAnn Mullally, Steven W Lane, Brian Ball, et al.Blood|March 15, 2013
Depletion of Jak2V617F myeloproliferative neoplasm-propagating stem cells by interferon-α in a murine model of polycythemia veraAnn Mullally, Claudia Bruedigam, Luke Poveromo, et al.Hematology. American Society of Hematology. Education Program|February 20, 2015
How does JAK2V617F contribute to the pathogenesis of myeloproliferative neoplasms?Edwin Chen, Ann MullallyPageof 41